岩石の脆性破壊のモデル化に関する研究

Microcracks-related fabric, determined using a stereological method, is represented quantitatively by means of the crack tensor introduced by, and brittle failure of granitic rock (Inada granite) is discussed based on the crack tensor analysis. Orientation of stress-induced microcracks doesn't change much during brittle failure, depending primarily on the orientation of pre-existing microcracks. Inelastic volumetric strain at failure decrease with increasing confining pressure due to the fact that crack opening is controlled by the confining pressure.textapplication/pdfdepartmental bulletin pape

Observation of B̅0→DsJ*(2317)+K- Decay

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Solar neutrino measurements in Super-Kamiokande-II

The results of the second phase of the Super-Kamiokande solar neutrino measurement are presented and compared to the first phase. The solar neutrino flux spectrum and time variation as well as oscillation results are statistically consistent with the first phase and do not show spectral distortion. The timedependent flux measurement of the combined first and second phases coincides with the full period of solar cycle 23 and shows no correlation with solar activity. The measured 8B total flux is (2:38± 0.05(stat.)/begin+0.16 // -0.15/end (sys.)) × 10^6 cm^{-2} s^{-1} and the day-night difference is found to be (-6.3 ±4.2(stat.)±3.7(sys.))%.There is no evidence of systematic tendencies between the first and second phases.journal articl

Evaluation Method of Thin Films in Consideration of Penetration Depth of Excitation Light by Raman Spectroscopy

Raman measurements was performed using two different wavelengths lasers for three different kinds of thickness GaAsN / GaAs thin film. We have compared two methods of fitting. One was fitted with TO and LO peak of sample and another was fitted with 3 peaks, TO, LO and the substrate GaAs LO peak. It was found that the signal from the substrate was superimposed on the signal from the upper layer. In the care of thin film Raman measurement, it is necessary either using the excitation light having the shorter penetration depth than the film thickness or analyzing process to eliminate the influence of the substrate.departmental bulletin pape

Modulation by Remember and Forget Cues

<p>Examples of remember and forget cues modulating neural activity in three delay neurons from three different birds. The cue and delay periods are shaded in grey. On remember trials, there is sustained activation in the cue and delay periods. On forget trials, the sustained activation is abolished. The binwidth is 50 ms. The vertical dashed lines separate the different periods of the task. ITI, intertrial interval; S, sample period.</p

Performance on Probe Trial Sessions

<div><p>(A) The performance on each of the eight sessions with forget-probe trials.</p> <p>(B) The performance on the probe sessions (±1 SE) averaged across all four birds. The dotted line represents chance levels of performance.</p></div

Histology

<div><p>(A) Lateral view of the pigeon brain. The NCL is shaded in red. The black line represents the intended electrode trajectory.</p> <p>(B) Histological reconstruction of the electrode tracks for the five pigeons. The NCL is shaded in red. The black lines represent the electrode track. All tracks were within the boundaries of the NCL. APH, area parahippocampalis; CDL, area corticoidea dorsolateralis; Hp, hippocampus; NC, nidopallium caudale; SGC, stratum griseum centrale; TrO: tractus opticus; V: ventricle.</p></div

The Response Profile of Excitatory Delay Neurons

<p>The response profile of all 102 instances of excitatory delay activity on remember and forget trials. To account for differences in firing rates between the neurons, each neuron's firing rate was normalized against its baseline firing rate. The cue and the delay periods are shaded in grey. The vertical dashed lines separate the different periods of the task. ITI, intertrial interval; S, sample period.</p

Behavioural Task

<p>Sequence of events on (A) remember trials, (B) forget trials, and (C) forget-probe trials. On remember and forget-probe trials the birds were presented with a test period, whereas on forget trials the test period was absent. The three horizontally arranged circles represent the projectors on which the stimuli, in this case a circle and dot, were displayed. During the cue and delay periods, the projectors were turned off. ITI, intertrial interval; R, remember cue, a high-frequency tone; F, forget cue, a low-frequency tone.</p

Assessment of genetic risk for myocardial infarction

application/pdfAlthough lifestyle and environmental factors influence the prevalence of myocardial infarction, genetic epidemiological studies have suggested that several genetic variants increase the risk for this condition. We have performed a large-scale association study to identify gene polymorphisms for reliable assessment of the genetic risk of myocardial infarction. The study population comprised 3,483 unrelated Japanese individuals (1,913 men; 1,570 women), including 1, 192 subjects with myocardial infarction and 2,291 controls. The genotypes for 164 polymorphisms of 137 candidate genes were determined with an oligonucleotide ligation assay based on analysis of fluorescent microspheres with suspension array technology. Multivariable logistic regression analysis with adjustment for age, sex, body mass index, and the prevalence of smoking, hypertension, diabetes mellitus, and hypercholesterolemia revealed that the 677C → T (Ala222Val) polymorphism of MTHFR, the 1595C → G (Ser447Stop) polymorphism of LPL, and the -108/3G → 4G polymorphism of IPF1 were significantly associated with the prevalence of myocardial infarction. A stepwise forward selection procedure demonstrated that IPF1, MTHFR, and LPL genotypes significantly affected the prevalence of myocardial infarction. Combined genotype analysis of these polymorphisms yielded a maximum odds ratio of 2.54 for the combined genotype of TT for MTHFR, CC for LPL, and 3G3G for IPF1. The genotypes for MTHFR, LPL, and IPF1 may prove reliable for assessment of genetic risk for myocardial infarction. Determination of the combined genotype for these genes may contribute to primary, personalized prevention of this condition.journal articl