A first measurement of the interaction cross section of the tau neutrino

The DONuT experiment collected data in 1997 and published first results in 2000 based on four observed $\nu_\tau$ charged-current (CC) interactions. The final analysis of the data collected in the experiment is presented in this paper, based on $3.6 \times 10^{17}$ protons on target using the 800 GeV Tevatron beam at Fermilab. The number of observed $\nu_\tau$ CC interactions is 9, from a total of 578 observed neutrino interactions. We calculated the energy-independent part of the tau-neutrino CC cross section ($\nu + \bar \nu$), relative to the well-known $\nu_e$ and $\nu_\mu$ cross sections. The ratio $\sigma(\nu_\tau)$/$\sigma(\nu_{e,\mu})$ was found to be $1.37\pm0.35\pm0.77$. The $\nu_\tau$ CC cross section was found to be $0.72 \pm 0.24\pm0.36 \times 10^{-38}$ cm$^{2}\rm{GeV}^{-1}$. Both results are in agreement the Standard Model.Comment: 37 pages, 15 figure

第3回先進軽量構造およびリフレクタアンテナに関する国際会議参加報告

application/pdf日本航空宇宙学会誌. 2019, 67 (6), P.228-229journal articl

最適に回収されたスラリーを用いたタングステンCMPの評価

Chemical Mechanical Polishing (CMP) is essential for the manufacturing of recent high-performance electronic device. However, for the CMP process, huge amount of slurry is used, which is to be disposed after being used only once. From the viewpoint of reduction of environmental burdens, this study was made for exploring the possibility of recycling of slurry by focusing on the slurry used for Tungsten CMP. For that purpose, we recovered the slurry used for polishing and then tried to reuse it for polishing again. As a method for evaluation, we made comparisons of removal rates between new slurry and recovered slurry. The results shows that when recovering the slurry during CMP, it contained the water remained on the surface of the polishing pad after dressing, by which slurry was diluted and caused the reduction of removal rate of Tungsten film. However, it was found that when making polishing of Tungsten film using the slurry recovered without being diluted with water, the same level of removal rate as new slurry is obtained.textapplication/pdfdepartmental bulletin pape

Study of Time-Dependent CP Violation in B0→J/ψπ0 Decays

journal articl

Influence of Inclination Angle of Polymer Material on Discharge Characteristics

Polymeric material has attracted much attention because of lightweight, high-insulation performance, and anti-weather aging performance. We have developed a polymer-housed arrester which can be useful for DC voltage application. In this study, we focused on relationship between inclination of polymer material and characteristics of the discharge ignition on the polymer surface. It was found that voltage stress and adhesion amount of water droplet on the polymer surface were important factors of occurrence of a continuous discharge.departmental bulletin pape

使える! 学べる!! 三重大図書館 : 学校図書館資源共有ネットワークにおける三重大学附属図書館の取組

application/pdfH.16-18学校図書館資源共有ネットワーク推進事業津 / 津市における研究実践成果報告会16学校図書館資源共有ネットワーク / これまでの取組 / 平成16年度:大学図書館として何ができるか模索 / 平成17年度:研修講座への参加、企画･実施 / 平成18年度:研修講座の企画･実施今後の連携に向けて / 三重大図書館を活用していただくために /conference objec

A Schematic of the Proposed Relationships of the Novel Immune Regulators, Sick and Dnr1, to Dredd and Rel

<p>Pointed and blunt arrows indicate activation and inhibition, respectively. Both Sick and Dredd are required for translocation of Rel to the nucleus and for activation of Dipt expression and they are consequently positioned upstream of Rel as activators. In the absence of Sick or Dredd, Dnr1 function is not needed to maintain pathway quiescence. Thus, Dnr1 is ordinarily required to either inhibit Sick and Dredd functions or to negate their actions, and we have indicated these regulators as being downstream of Dnr1 (A). Although we have no epistatic data that separates the action of Sick and Dredd, Dredd appears to directly cleave Rel and is hence likely to be immediately upstream of Rel. Sick might function in conjunction with Dredd or as an activator of Dredd. Since <i>dnr1</i> RNAi does not enhance the response to LPS, we suggest that its inhibitor activity is either repressed or bypassed upon exposure to LPS. Consequently, we have shown that treatment with LPS counteracts Dnr1-dependent Dredd inhibition (B). We do not mean to preclude other actions of LPS that might contribute to induction, but it is notable that inactivation of Dnr1 is sufficient to activate signaling. Finally, we have shown that Dnr1 levels are affected by Dredd, and we have indicated this with a positive feedback arrow.</p

List of Modulators of the Immune Response and a False Color Display of Their Influence on Dipt-lacZ Induction

<p>The genes identified as DDRi (A), EDRi (B), and CDRi (C) are listed, and the colored bars show the influence of the corresponding dsRNA on Dipt-lacZ expression. The top two entries in (A), (B), and (C) show control cells (no dsRNA) without and with LPS, respectively. The scales for the false colors are given at the bottom left. Dipt-lacZ levels are given in terms of percent positive cells. For exact Dipt-lacZ expression values for each dsRNA refer to accompanying supplemental tables. In (B), the color scale (right) is compressed and extended compared to (A), and an asterisk indicates genes that also caused a CDRi phenotype. In (C), the pound sign indicates morphological defects and an asterisk indicates genes that also caused an EDRi phenotype, and the division of the genes into epistatic groups is shown. To the immediate left a false-color bar (coded as in [B]) indicates the effect of the dsRNAs on Dipt-lacZ expression without LPS addition. The block of colored columns shows the results of epistasis tests. Here, we set the undisturbed level of CDRi activation to 100% (as indicated in the left column in this group and the color code below), and to the right we represent reduction of this activation by prior RNAi of different Imd pathway genes. Five epistatic clusters (I–V) were identified (indicated by the lines to the left).</p

A GFP-Relish Reporter Cell Line Subdivides DDRi dsRNA into Three Categories

<div><p>(A–B) Immunofluorescence of GFP-Relish cells with GFP-Relish in green, DNA in blue, and actin in red. Relish is predominantly cytoplasmic in untreated control cells and rapidly translocates to the nucleus of cells incubated with LPS.</p> <p>(C) An anti-GFP Western blot of lysates harvested from GFP-Relish cells treated with LPS for different periods. GFP-Relish rapidly shifts from a full-length form to a shorter processed form after exposure to LPS, and full-length Relish gradually reaccumulates.</p> <p>(D–E) Immunohistochemistry of GFP-Relish cells incubated with GFP-expressing E. coli (arrowheads) and treated with (E) or without (D) dsRNA against PGRP-LC. Imd pathway inactivation prevents bacterial-induced Relish nuclear translocation.</p> <p>(F) Shows effects of treatment of GFP-Relish cells with DDRi dsRNAs for 4 d prior to LPS treatment. GFP-Relish was scored as cytoplasmic (uninduced), nuclear (induced), or reduced in amount (abnormal).</p> <p>(G) Shows an epistatic analysis of the DDRi, <i>sick.</i> Suppression of <i>sick</i> interferes with Dipt-lacZ induction by Group III, IV, and V CDRi dsRNAs, but not those of Groups I and II, suggesting that Sick acts downstream of Imd and Dredd, but upstream of Relish in signal transduction.</p></div