One-neutron halo structure in C-15

The one- or two-neutron removal reactions as well as reaction cross sections for C-14,C-15 on carbon target have been studied by using 110A MeV Ne-22 primary beam on Riken Projectile Fragment Separator in RIKEN. The longitudinal momentum distributions of C-13,C-14 fragments from C-15 and C-13 fragments from C-14 breakup have been measured at 83A MeV by means of direct time-of-flight method. Full width at half maximum (FWHM) of the distributions have been determined to be 71+/-9 MeV/c and 223+/-28 MeV/c for C-14 and C-13 from C-15, and 195+/-21 MeV/c for C-13 from C-14. The FWHM for C-13 fragments from C-15 and C-14 breakup are consistent with the Goldhaber model's prediction. While the FWHM of C-14 fragments from C-15 is much smaller, which confirms the experimental results from MSU and GANIL, an anomalous enhancement from its neighbors has been observed in the measured reaction cross section of C-15. The experimental data are discussed in the framework of the Glauber model. The analysis of both the fragment momentum distributions and reaction cross sections indicates a dominant s-wave component in the ground state of C-15.Copyright notice(c)2004 American Physical Society. All rights reserved. Publisher's version: http://prola.aps.org/abstract/PRC/v69/i3/e034613textapplication/pdfjournal articl

Observation of B+→ψ(3770)K+

journal articl

Measurement of branching fraction and time-dependent CP asymmetry parameters in B^0 → D^*+D^*-K^{0}_{S}decays

We present a measurement of the branching fraction and time-dependent CP violation parameters for B^0 → D^*+D^*-K^{0}_{S} decays. These results are obtained from a 414 fb^{-1} data sample that contains 449×10^6 B\bar{B} pairs collected at the Υ(4S) resonance with the Belle detector at the KEKB asymmetric-energy e^+e^- collider. We obtain the branching fraction, B(B^0→D^*+D*^-K^{O}_{S})= [3.4±0.4(stat)±0.7(syst)]×10^-3, which is in agreement with the current world average. We also obtain an upper limit on the product branching fraction for a possible two-body decay, B(B^0 → D_s1^+(2536)D^*-)B(D_s1^+(2536)→ D^*+K^{O}_{S})s^+, where s^±[equivalent]m2(D^*±K^{0}_{S}), we extract the CP violation parameters, J_c/J_0=0.60^{+0.25}_{-0.28}(stat)±0.08(syst), 2J_s1/J_0sin2φ_1=-0.17^{+0.42}_{-0.42}(stat)±0.09(syst), 2J_s2/J_0cos2φ_1=-0.23^{+0.43}_{-0.41}(stat)±0.13(syst). A large value of J_c/J_0 would indicate a significant resonant contribution from a broad unknown D_s^**+ state. Although the sign of the factor, 2J_s2/J_0, can be deduced from theory, no conclusion can be drawn regarding the sign of cos2φ_1 given the errors.journal articl

2020ネンド トショカン ゲンバ エンシュウ ホウコク アカシ シリツ アカシ シミン トショカン イイダ シリツ チュウオウ トショカン イバラキ シリツ チュウオウ トショカン ウジシ チュウオウ トショカン オーテピア コウチ トショカン オオサカ シリツ チュウオウ トショカン オオサカ フリツ ダンジョ キョウドウ サンカク セイショウネン センター ジョウホウ ライブラリー オオサカ フリツ チュウオウ トショカン オオサカ フリツ ナカノシマ トショカン キョウトシ ウキョウ チュウオウ トショカン キョウトシ チュウオウ トショカン キョウトシ フシミ チュウオウ トショカン キョウト フリツ キョウトガク レキサイカン キョウト フリツ トショカン クマモト シリツ トショカン コウベ シリツ チュウオウ トショカン コクリツ コッカイ トショカン トウキョウ ホンカン ジョウヨウ シリツ トショカン タカツキ シリツ チュウオウ トショカン タカラズカ シリツ トショカン ツシマ シリツ トショカン ドウシシャ コウトウ ガッコウ トショカン ドウシシャ ジョシ ダイガク トショ ジョウホウ センター ドウシシャ ジョシ チュウガッコウ コウトウ ガッコウ トショ ジョウホウ センター ドウシシャ ダイガク トショカン トクシマ シリツ トショカン トヨナカ シリツ オカマチ トショカン ナラ ケンリツ トショ ジョウホウカン ハンナン シリツ トショカン ヒラカタ シリツ チュウオウ トショカン フクチヤマ シリツ トショカン ヤス シリツ トショカン ヤマトコオリヤマ シリツ トショカン ヤワタ シリツ トショカン

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生研公開講座 : トルコ, そして台湾の地震が突きつけた課題

departmental bulletin pape

(a)The pipeline used for the annotation of the kinase genes is represented in this schema, information deriving from the gene annotations are recorded into the relational database KinWeb

<p><b>Copyright information:</b></p><p>Taken from "Systematic analysis of human kinase genes: a large number of genes and alternative splicing events result in functional and structural diversity"</p><p></p><p>BMC Bioinformatics 2005;6(Suppl 4):S20-S20.</p><p>Published online 1 Dec 2005</p><p>PMCID:PMC1866387.</p><p></p> (b) Count of domains identified within the kinase gene pool.(c)Prediction of structure features in kinase protein sequences

(a) The "Gene Report" page shows gene annotations extracted from public databases, the results of predictions for transmembrane domains and Cys disulfide bonding state and links the identified CSTs to the graphical representation of the gene

<p><b>Copyright information:</b></p><p>Taken from "Systematic analysis of human kinase genes: a large number of genes and alternative splicing events result in functional and structural diversity"</p><p></p><p>BMC Bioinformatics 2005;6(Suppl 4):S20-S20.</p><p>Published online 1 Dec 2005</p><p>PMCID:PMC1866387.</p><p></p> (b) Graphical representation of the gene and related transcripts and CSTs. Details are shown for the CTS under the mouse pointer. CSTs are color-coded based on degree of conservation. (c) The CST details page, where full details of the selected CST are shown, including the results of BLAST searches

The interface provides access to the data in various ways: (a) The KinWeb home page

<p><b>Copyright information:</b></p><p>Taken from "Systematic analysis of human kinase genes: a large number of genes and alternative splicing events result in functional and structural diversity"</p><p></p><p>BMC Bioinformatics 2005;6(Suppl 4):S20-S20.</p><p>Published online 1 Dec 2005</p><p>PMCID:PMC1866387.</p><p></p> A query form allows to search by gene name, classification group or domain. (b) The genome browser, allowing view and selection of the genomic regions where kinase genes are located. (c) The multi-domain search page. Here kinases can be searched according to a combination of domains. (d) Example of results from multi-domain search in (c). For each protein, all domains predicted by InterProScan are graphically represented

Systematic analysis of human kinase genes: a large number of genes and alternative splicing events result in functional and structural diversity-0

<p><b>Copyright information:</b></p><p>Taken from "Systematic analysis of human kinase genes: a large number of genes and alternative splicing events result in functional and structural diversity"</p><p></p><p>BMC Bioinformatics 2005;6(Suppl 4):S20-S20.</p><p>Published online 1 Dec 2005</p><p>PMCID:PMC1866387.</p><p></p>indicated as when completely included in one exon, or when overlapping the 5' or 3' end of one exon, when CST contains one exon, or when a larger CST covers more than one exon of one or more transcripts. (b) CSTs contained within the analysed kinase genes: 20% of the CSTs, initially annotated as intronic, are indicated as "exon-like" because positive to one or more of the tests described under methods. (c) Exon-like CSTs within the analysed kinase genes, positive to one, two, three and four of the used test criteria (see methods). (d) Number of kinase genes containing at least one exon-like CST positive to two, three or four tests