Search for B decays into η'p, η'K^*, η'φ, η'ω and η'η^(')

We report on a search for the exclusive two-body charmless hadronic B meson decays B → η'ρ, B → η'K^*, B^0 → ηφ, B^0 → η'ω, and B0 →η'η^('). The results are obtained from a data sample containing 535×10^6 B\bar{B} pairs that were collected at the Υ(4S) resonance with the Belle detector at the KEKB asymmetric-energy e^+e^- collider. We find no significant signals and report upper limits in the range (0.5–6.5)×10^-6 for all of the above decays.journal articl

Observation of Large CP Violation and Evidence for Direct CP Violation in B0→π+π- Decays

journal articl

The Study of Staff's View about Children in Children's Homes : The Use of Focus Interview

departmental bulletin pape

論説 : エネルギー・熱工学とサステナビリティー : エネルギー資源, 地球環境そしてサステナビリティに係わる課題

departmental bulletin pape

Observation of Size-Dependent Optical Properties Based on Surface and Quantum Effects in Nanocrystals of 5,5′-Bis(4-Biphenylyl)-2,2′-Bithiophene

Herein, 5,5'-bis(4-biphenylyl)-2,2'-bithiophene (BP2T or PPTTPP) nanocrystals are prepared via a modified miniemulsion technique. X-ray diffraction and selected area electron diffraction analyses reveal that the BP2T molecules stand almost upright against the crystal basal plane. With decreasing nanocrystal size, the peak energies of photoluminescence (PL) spectra are blue-shifted in the photon energy range from the bulk crystal to the monomer state. Energy-wavevector dispersion plots reproduced using the data of size-dependent PL spectra indicate that the size dependence of optical properties cannot be explained by the conventional quantum confinement effect used in inorganic nanocrystals. The micro-PL and time-resolved PL measurements of the BP2T nanocrystals reveal that the size-dependent optical properties can be attributed to a combined effect based on the surface effects as well as lattice softening, and quantum effects involving specific surface area-dependent site shift effect and exciton confinement. Moreover, amplified spontaneous emission is observed from the nanocrystals ?600-1000 nm in size, which is smaller than the size previously reported for organic systems.journal articl

A mode-in-state contribution factor based on Koopman operator and its application to power system analysis

application/pdfNonlinear Theory and Its Applications. 2022, 13 (2), P.409-414journal articl

The downregulation of PPARγ expression by FFAs requires TLR4dependent activation of the ER stress.

<p>3T3L1 adipocytes were pretreated with neutralizing antibodies to TLR4 (A, B), or control IgG prior to FA treatment (500 µM, 3 hr). Adipocyte PPARγ protein content was assessed as above, by immunoblotting analysis (A) and mRNA by real time qPCR (B). C) Mature 3T3L1 were treated with tunicamycin (TUN, 1 µg/ml) and thapsigargin (TG, 50 nM) for the indicated times prior to extract RNA and quantify PPARγ, CHOP-10 and KC with qPCR. D) Mature 3T3L1 adipocytes were pretreated 3 hours with 14-phenyl butyrate (PBA, 10 mM) and tauroursodeoxycholic acid (TUDCA, 500 µg/ml) prior to be exposed to ARA (100 µM) for 6 hours. Proteins were then extracted and PPARγ was quantified by western-blot. E) Mature 3T3L1 adipocytes were pretreated 3 hours with 14-phenyl butyrate (PBA, 10 mM) before exposure to 100 µM ARA. PPARγ expression was quantified with qPCR. ** P<0.01.</p

Arachidonic Acid prevents PPARγ transrepressional activity on chemokines secretion by adipocytes.

<p>MCP1 concentration was quantified with ELISA from 24 hours 3T3L1 conditioned media (A). Conditioned media were either prepared from adipocytes not stimulated (Veh), treated with TNFα (10 ng/ml), with 500 µM of FFAs mixture (containing 100 µM Arachidonic Acid) or with 100 µM of Arachidonic Acid (ARA) alone. Cotreatment with Rosi (1 µM) was also perfomed (white bars). Media used for preparation of conditioned media is shown for reference (−). QPCR analysis of PPARγ (B) MCP1 and KC (C) and adiponectin (AdipoQ) and aquaporin 7 (AQP7) (D) expression in 3T3L1 adipocytes exposed to 100 µM of Arachidonic Acid (ARA), Rosi (1 µM) or both compounds; ** p<0.01 <i>vs</i> Untreated, # p<0.05 <i>vs</i> Rosi. Letters above the bars show statistical groups (p<0.05).</p

PPARγ signaling decreases secretion of chemoattractants from adipocytes.

<p>A) Mature 3T3L1 adipocytes were exposed 24 hours to 500 µM FFA mixture (arachidonic, lauric, linoleic, myristic and oleic acids, 100 µM final each) in presence or absence of Rosi (1 µM). After washes, 24 hours adipocytes conditioned media were prepared, in absence of treatment and used for chemotaxis assay on Raw264.7 macrophages. B) PPARγ siRNA or controls (Ctrl) were injected in mature 3T3L1 adipocytes. 24 hours after electroporation, adipocytes were exposed to FFA mixture or vehicle, for 16 hours. After washes, 24 hours adipocytes conditioned media were prepared, in absence of treatment and used for chemotaxis assay on Raw264.7 macrophages. C) QPCR was used to quantify PPARγ and MCP1 from the adipocytes treated as above. D) Western-blots were performed in order to quantify efficient PPARγ knockdown, HSP90 protein is shown as internal control. * P<0.05; # P<0.05 for Rosi <i>vs</i> Vehicle in absence of FFA in 1A; and for PPARγ siRNA <i>vs</i> Luciferase siRNA in absence of FFA.</p

Rosiglitazone decreases chemokine expression in adipocytes and chemokine receptor expression in SVCs.

<p>Expression of macrophages chemokines in adipocytes fraction (A) and corresponding receptors (B) in stromal vascular fraction were quantified with QPCR. Eotaxin-1 and corresponding receptor, CCR3 are shown in white, MIP1α and RANTES corresponding receptors CCR1 and CCR5 are shown in light grey, Fractalkine and corresponding receptor CX3CR1 are shown in dark grey, MCP1 and corresponding receptor, CCR2, are shown in black. C) QPCR analysis of PPARγ in adipocytes and SV fractions. D) TNFα expression in stromal vascular fraction was determined by qPCR. D) CD68 and Glut4 expression were quantified from SVF and adipocytes fractions by qPCR. ** P<0.01. A, B above the bars show statistical groups (p<0.05).</p