日本人の平和認識 / 学派の「多元主義」「連帯主義」論争に照らして

departmental bulletin pape

An Improved Method of “CloneSpot” for Detecting Repackaged Applications Using Metadata

Since Android is widely used as an operating system (OS) for smartphones, applications running on the OS are being targeted by malware developers. Android applications are generally created using Java, which means it is easy to decompile the application and add or change the code; therefore, malware developers can easily insert malware and illegal advertisements into legitimate applications. The modified applications are called repackaged applications. These applications are uploaded to a market for users to install, however since repackaged applications have a negative effect on users, their detection and removal are important. The Developers of repackaged applications tend to be reluctant to make large changes to data, called application metadata, to make users misrecognition. To detect repackaged applications, “CloneSpot” has been proposed which uses the above trends, however, improvements are required for efficient detection. In this study, we propose an improved method of “CloneSpot” for the efficient detection of repackaged applications. Specifically, we improve the clustering of “CloneSpot” and introduce evaluation indexes for efficient repackaged application detection. In the evaluation experiments, we evaluated the clustering accuracy and the repackaged application estimation accuracy of the proposed method.departmental bulletin pape

Observation of B̅0→D(*)0pp̅

journal articl

第2章 数学的リテラシーから考える授業研究(VIII. 公開授業・研究会)

2009-01-15国立情報学研究所で電子化したコンテンツを使用している。departmental bulletin pape

Block-copolymers, nanocomposites, organic/inorganic hybrids, polymethylenes.

Includes bibliographical references and index.Book fair 2013.ix, 235 p. :This book contains short and concise reports on physics and chemistry of polymers, each written by the world renowned experts. The book has the Highest Impact Factor of all publications ranked by ISI within Polymer Science

Effects of blocking eCB reuptake on sub threshold tetanus.

<p>Averaged time courses of the experiments in which the 5 min at 10 Hz protocol was given in control ACSF (open circles) of after pre-treatment with the eCB reuptake blocker AM404 (20 µM, black circles).</p

Inhibition of DGL-α, the enzyme that synthesizes 2-AG, blocked eCB-LTD.

<p>Averaged time courses of the experiments in which the 10 min at 10 Hz protocol was given in control ACSF (open circles) of after pre-treatment with tetrahydrolipstatin (THL, 10 µM, black circles), an inhibitor of the DGL-α.</p

Role of 2-AG in eCB-LTD LTD in the PrPFC.

<p>(A) Typical experiment showing that a 5 min stimulation at 10 Hz is sub threshold to induce LTD, even when applied two consecutives times. Calibration bars: x: 10 ms, y: 0.2 mV. (B) Representative experiment showing that 5 min at 10 Hz can induce LTD when applied after bath perfusion with URB 602 (100 µM). Traces were taken at the time indicated on corresponding graph. Calibration bars: x: 10 ms, y: 0.2 mV. (C) Summary bar histogram of all the experiments performed where the first tetanus was given in saline and the second tetanus was given after bath perfusion of URB602. LTD was induced only when URB was present (t-test, p = 0.0271). (D) Averaged time courses of the experiments in which the 5min at 10Hz protocol was given in control ACSF (open circles) of after pre-treatment with URB597 (2 µM, black triangles) or URB602 (black circles).</p

Immunocytochemical localization of mGluR5/DGL-α (A, B) and CB1R/ DGL-α (C, D) in mouse prPFC cortical layers V/VI.

<p>Double preembedding immunogold and immunoperoxidase methods for electron microscopy. (A, B) mGluR5 metal particles (arrows) and DGL-α immunodeposits colocalized in postsynaptic dendrites (den) and dendritic spines (s). mGluR5 labeling was in perisynaptic and extrasynaptic membranes. No mGluR5/DGL-α immunoreactivity was observed in presynaptic terminals (T). (C, D) CB1R immunoparticles were on presynaptic terminals membranes (T) away from synaptic specializations made on postsynaptic DGL-α-immunoreactive dendritic spines (s). Observe that DGL-α-positive spines also received CB1R-immunonegative synaptic terminals, and that a CB1R-labeled presynaptic terminal (thick arrow) probably of inhibitory nature (IT in D) made a synapse with a postsynaptic DGL-α-negative dendritic branchlet. Scale bars: 0.5 µm.</p

Postsynaptic receptors and transduction pathways involved in eCB-LTD.

<p>(A) eCB-LTD was not affected by a mixture of the NMDAR antagonist MK801 (40 µM), the D1 receptor antagonist SCH23390 (25 µM) and the D2 receptor antagonist sulpiride (25 µM). (B) The mGluR5 antagonist MPEP (10 µM) completely blocked eCB-LTD (C) Bar graph summarizing experiments showing that the non subtype selective group 1mGluR antagonist LY341495 (50 µM) and the Phospholipase C inhibitor U73122 both prevented eCB-LTD induction. 45 min after the end of the tetanus, the fEPSPs was 76.6±2.64% (n = 62) of baseline in control and 95.8±4.85% (n = 12, p = 0.004 t-test) and 96.33±6.4% (n = 11, p = 0.0009 t-test) in LY341495, respectively. (D) eCB-LTD requires postsynaptic Ca²⁺ rise. Time course of all the experiments performed where the recording pipette was filled with BAPTA (20 mM, n = 11) and where eCB-LTD was completely blocked.</p