546 research outputs found

    Search for the standard model Higgs boson at LEP

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    Search for rare quark-annihilation decays, B --> Ds(*) Phi

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    We report on searches for B- --> Ds- Phi and B- --> Ds*- Phi. In the context of the Standard Model, these decays are expected to be highly suppressed since they proceed through annihilation of the b and u-bar quarks in the B- meson. Our results are based on 234 million Upsilon(4S) --> B Bbar decays collected with the BABAR detector at SLAC. We find no evidence for these decays, and we set Bayesian 90% confidence level upper limits on the branching fractions BF(B- --> Ds- Phi) Ds*- Phi)<1.2x10^(-5). These results are consistent with Standard Model expectations.Comment: 8 pages, 3 postscript figues, submitted to Phys. Rev. D (Rapid Communications

    dOCRL maintains immune cell quiescence in Drosophila by regulating endosomal traffic

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    Lowe Syndrome is a developmental disorder characterized by eye, kidney, and neurological pathologies, and is caused by mutations in the phosphatidylinositol-5-phosphatase OCRL. OCRL plays diverse roles in endocytic and endolysosomal trafficking, cytokinesis, and ciliogenesis, but it is unclear which of these cellular functions underlie specific patient symptoms. Here, we show that mutation of Drosophila OCRL causes cell-autonomous activation of hemocytes, which are macrophage-like cells of the innate immune system. Among many cell biological defects that we identified in docrl mutant hemocytes, we pinpointed the cause of innate immune cell activation to reduced Rab11-dependent recycling traffic and concomitantly increased Rab7-dependent late endosome traffic. Loss of docrl amplifies multiple immune-relevant signals, including Toll, Jun kinase, and STAT, and leads to Rab11-sensitive mis-sorting and excessive secretion of the Toll ligand Spåtzle. Thus, docrl regulation of endosomal traffic maintains hemocytes in a poised, but quiescent state, suggesting mechanisms by which endosomal misregulation of signaling may contribute to symptoms of Lowe syndrome

    Observation of the Decay B=> J/psi eta K and Search for X(3872)=> J/psi eta

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    We report the observation of the BB meson decay B±J/ψηK±B^\pm\to J/\psi \eta K^\pm and evidence for the decay B0J/ψηKS0B^0\to J/\psi \eta K^0_S, using {90} million BBbarBBbar events collected at the \ensuremath{\Upsilon{(4S)}}\xspace resonance with the BaBarBaBar detector at the PEP-II e+ee^+ e^- asymmetric-energy storage ring. We obtain branching fractions of B\cal{B}(B±J/ψηK±(B^\pm\to J/\psi \eta K^{\pm})=(10.8±2.3(stat.)±2.4(syst.))×105(10.8\pm 2.3(\rm{stat.})\pm 2.4(\rm{syst.}))\times 10^{-5} and B\cal{B}(B0J/ψηKS0(B^0\to J/\psi\eta K_{\rm{S}}^{0})=(8.4±2.6(stat.)±2.7(syst.))×105(8.4\pm 2.6(\rm{stat.})\pm 2.7(\rm{syst.}))\times 10^{-5}. We search for the new narrow mass state, the X(3872), recently reported by the Belle Collaboration, in the decay B^\pm\to X(3872)K^\pm, X(3872)\to \jpsi \eta and determine an upper limit of B\cal{B}(B^\pm \to X(3872) K^\pm \to \jpsi \eta K^\pm) <7.7×106<7.7\times 10^{-6} at 90% C.L.Comment: 7 pages and two figures, submitted to Phys. Rev. Lett

    Measurement of branching fractions and mass spectra of B -> K pi pi gamma (vol 98, art no 211804, 2007)

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    Contains fulltext : 71970.pdf (publisher's version ) (Open Access)2 p

    Search for the rare leptonic decay B-->tau(-)nu(tau)

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    We present a search for the decay B- -> tau(-)(tau) in a sample of 88.9 x 10(6) B (B) over bar pairs recorded with the BABAR detector at the Stanford Linear Accelerator Center B factory. One of the two B mesons from the Upsilon(4S) is reconstructed in a hadronic or a semileptonic final state, and the decay products of the other B in the event are analyzed for consistency with a B- -> tau(-)(tau) decay. We find no evidence of a signal and set an upper limit on the branching fraction of B((B) over bar -> tau(-)(tau)) < 4.2 x 10(-4) at the 90% confidence level

    Measurement of branching fractions and mass spectra of B -> K pi pi gamma

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    We present a measurement of the partial branching fractions and mass spectra of the exclusive radiative penguin processes B -> K pi pi gamma in the range m(K pi pi)pi(+)pi(-). Using 232x10(6) e(+)e(-)-> B (B) over bar events recorded by the BABAR experiment at the SLAC PEP-II asymmetric-energy storage ring, we measure the branching fractions B(B+-> K+pi(-)pi(+)gamma)=[2.95 +/- 0.13(stat)+/- 0.20(syst)]x10(-5), B(B-0 -> K+pi(-)pi(0)gamma)=[4.07 +/- 0.22(stat)+/- 0.31(syst)]x10(-5), B(B-0 -> K-0 pi(+)pi(-)gamma)=[1.85 +/- 0.21(stat)+/- 0.12(syst)]x10(-5), and B(B+-> K-0 pi(+)pi(0)gamma)=[4.56 +/- 0.42(stat)+/- 0.31(syst)]x10(-5)

    Measurements of the branching fraction and CP-violation asymmetries in B-0 -> f(0)(980)K-S(0)

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    We present measurements of the branching fraction and CP-violating asymmetries in the decay B-0-->f(0)(980)K-S(0). The results are obtained from a data sample of 123x10(6) Y(4S)-->B (B) over bar decays. From a time-dependent maximum likelihood fit, we measure the branching fraction B(B-0-->f(0)(980)(-->pi(+)pi(-))K-0)=(6.0+/-0.9+/-0.6+/-1.2)x10(-6), the mixing-induced CP violation parameter S=-1.62(-0.51)(+0.56)+/-0.09+/-0.04, and the direct CP violation parameter C=0.27+/-0.36+/-0.10+/-0.07, where the first errors are statistical, the second systematic, and the third due to model uncertainties. We measure the f(0)(980) mass and width to be m(f0)(980)=(980.6+/-4.1+/-0.5+/-4.0) MeV/c(2) and Gamma(f0)(980)=(43(-9)(+12)+/-3+/-9) MeV/c(2), respectively
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