Gastroesophageal intussusception with complete herniation of the spleen in a 12 months old dog with idiopathic megaoesophagus

A 12 months old, castrated male, mix-breed dog was presented due to acute onset of vomiting, retching, anorexia, and tachypnoea. Idiopathic megaoesophagus was diagnosed three months prior to presentation. Radiographic and CT examination revealed gastroesophageal intussusception with herniation of the complete spleen into the intussusception. After initial stabilization surgical treatment was performed. The stomach and spleen were manually reduced into the abdomen. Due to questionable viability of the gastric wall an inverting suture pattern was used to invaginate the compromised part. Left sided gastropexy was performed to reduce risk of recurrence. Additionally oesophagopexy was performed to reduce the risk of hiatal hernia due to intraoperative damage to the hiatus. The patient recovered uneventful and was discharged from hospital five days following surgery. Conservative treatment of concurrent megaoesophagus was continued. At last follow up, 10 months later, the dog was clinically fine, had gained weight, and showed no signs of regurgitation

Pylorogastric intussusception in the dog: a case report and literature review

A 10-month-old, neutered male Saint Bernard presented for evaluation of acute, severe vomiting. A soft-tissue mass was noted within the stomach on survey abdominal radiographs. The diagnosis of pylorogastric intussusception was made during exploratory celiotomy. The intussusception was manually reduced at surgery, the pyloric antrum was enlarged, and the duodenum was permanently affixed to the abdominal wall in an attempt to prevent recurrence of the intussusception. The dog recovered, has gained weight (5 kg), and has had only one isolated episode of vomiting during the one year since discharge from the hospital. This report documents the fourth reported case of pylorogastric (i.e., duodenogastric, gastrogastric) intussusception in the veterinary literature and is the first report that details the surgical management of the disease.</jats:p

Potential central nervous system complications of von Willebrand's disease

Three Doberman pinschers were presented on emergency referral for progressive neurological deficits. All three dogs had a similar onset of clinical signs associated with an apparently minor traumatic event. Each dog progressed to significant neurological dysfunction including paraplegia, tetraplegia, and/or loss of deep pain sensation. None of the animals was apparently affected by cervical vertebral instability ("Wobbler's Syndrome"). All were confirmed to have von Willebrand's disease. In all cases, significant epidural hemorrhage was identified. The etiology of each hemorrhage, however, was different for each animal. The cases presented here demonstrate a potential relationship between neurological deficits and the patient's ability to effectively coagulate blood. Hemostatic abnormalities, such as von Willebrand's disease, should be included as possible differential diagnoses or contributing factors in animals demonstrating neurological deficits. These abnormalities should especially be considered following trauma, intervertebral disk extrusion, or spinal surgery.</jats:p