Electrooxidation of formic acid on gold : An ATR-SEIRAS study of the role of adsorbed formate

Funding from the DGI (Spanish Ministry of Education and Science) through Projects CTQ2009-07017 and PLE2009-0008 is gratefully acknowledged. M.E.-E. acknowledges an FPI fellowship from the Spanish Ministry of Science and Innovation and an accommodation grant at the Residencia de Estudiantes from the Madrid City Council. C. V.-D. acknowledges a JAE-Doc fellowship from CSIC.Peer reviewedPostprin

パリはパリを擬態する -オリンピック都市開発とグラン・パリ-

https://www.newsweekjapan.jp/asteion/magazine/vol101/departmental bulletin pape

Geographical Research for the Protection of the Human Rights of Irregular Migrants -Research Trends in Japan and the Current Situation in Europe-

departmental bulletin pape

The Chromatin Modifier MSK1/2 Suppresses Endocrine Cell Fates during Mouse Pancreatic Development

Type I diabetes is caused by loss of insulin-secreting beta cells. To identify novel, pharmacologically-targetable histone-modifying proteins that enhance beta cell production from pancreatic progenitors, we performed a screen for histone modifications induced by signal transduction pathways at key pancreatic genes. The screen led us to investigate the temporal dynamics of ser-28 phosphorylated histone H3 (H3S28ph) and its upstream kinases, MSK1 and MSK2 (MSK1/2). H3S28ph and MSK1/2 were enriched at the key endocrine and acinar promoters in E12.5 multipotent pancreatic progenitors. Pharmacological inhibition of MSK1/2 in embryonic pancreatic explants promoted the specification of endocrine fates, including the beta-cell lineage, while depleting acinar fates. Germline knockout of both Msk isoforms caused enhancement of alpha cells and a reduction in acinar differentiation, while monoallelic loss of Msk1 promoted beta cell mass. Our screen of chromatin state dynamics can be applied to other developmental contexts to reveal new pathways and approaches to modulate cell fates

On the Resistance between Main Electrodes on the Perimeter of the Two-Dimensional Domain

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Relations among Moduli for obtaining Resistances between Electrodes Variously Arranged on the Perimeter of the Two-dimensional Domain

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Exact Resistance Values between two Electrodes attached specially on the Perimeter of the Domain composed of some particular Polygons

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Calculation of the Resistance Value between two Electrodes attached on the Periphery of a Resistance Plate

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