An in-depth look at prior art in fast round-robin arbiter circuits

Arbiters are found where shared resources exist such as busses, switching fabrics, processing elements. Round-robin is a fair arbitration method, where requestors get near-equal shares of a common resource or service. Round-robin arbitration (RRA) finds use in network switches/routers and processor boards/systems as well as many other applications that have concurrency. Today's electronic systems require arbiters with hundreds of ports (e.g., switching fabrics with virtual I/O queues) and clock speeds near the limits of even the latest microelectronics fabrication processes/libraries. Achieving high clock speeds in the presence of large number of ports is only possible with highly parallel arbiter architectures. This paper presents an in-depth literature survey of previous work on this problem. It looks at RRA work in the literature in a bigger context, then defines the typical RRA problem (RRA_typical), and specifically investigates work on fast architectures that solve the RRA_typical problem. There are five such works that are really competitive. This report takes a very in-depth look at these works. It explains each architecture and how/why it works from a unique perspective that cannot be found in the original publication of that architecture. It also proposes improvements to these architectures. We wrote generators for the improved versions of these architectures. We will share a summary of synthesis results in this report – although a detailed account of how these results were obtained and their analysis is the subject of another (upcoming) publicatio

The free growth criterion for grain initiation in TiB

γ-titanium aluminide (γ-TiAl) based alloys enable for the design of light-weight and high-temperature resistant engine components. This work centers on a numerical study of the condition for grain initiation during solidification of TiB2 inoculated γ-TiAl based alloys. Grain initiation is treated according to the so-called free growth criterion. This means that the free growth barrier for grain initiation is determined by the maximum interfacial mean curvature between a nucleus and the melt. The strategy presented in this paper relies on iteratively increasing the volume of a nucleus, which partially wets a hexagonal TiB2 crystal, minimizing the interfacial energy and calculating the corresponding interfacial curvature. The hereby obtained maximum curvature yields a scaling relation between the size of TiB2 crystals and the free growth barrier. Comparison to a prototypical TiB2 crystal in an as cast γ-TiAl based alloy allowed then to predict the free growth barrier prevailing under experimental conditions. The validity of the free growth criterion is discussed by an interfacial energy criterion

Lymph node (but not spleen) invasion by murine lymphoma is both CD44- and hyaluronate-dependent.

Abstract Similar to activated T cells, LB T cell lymphoma expresses the CD44 cell surface Ag. In addition, the vast majority of LB cells also express the beta 2 (CD18) and alpha L (CD11a) chains of LFA-1 integrin. In view of the finding that anti-CD18 mAb blocked spleen, but not lymph node invasion by LB cells inoculated s.c. into BALB/c mice, we tested the ability of anti-CD44 mAb (IM 7.8.1) to block the infiltration of LB cells into the lymph nodes. We found that, as opposed to anti-CD18 mAb, anti-CD44 mAb, as well as its F(ab')2 or Fab fragment, prevented lymph node infiltration but had no effect on spleen invasion. This conclusion was based on histologic examination and [3H]thymidine incorporation into proliferating LB cells invading the lymphoid organs. Histologic analysis further demonstrated that LB cells invade the lymph node via the afferent lymphatics. The surface expression of CD44 molecules on LB cells was enhanced after PMA activation. PMA activation also enabled in vitro binding of the lymphoma to hyaluronic acid (HA), a known ligand of CD44. Because anti-CD44 mAb, its F(ab')2 or Fab fragment, and hyaluronidase blocked this binding, we also tested the ability of the enzyme to inhibit lymph node invasion by LB cells. We established through histologic examination and [3H]thymidine incorporation that hyaluronidase protected the lymph node, but not the spleen, from invasion by the lymphoma.</jats:p