22 research outputs found
Identification and Lossy Reconstruction in Noisy Databases
A high-dimensional database system is studied where the noisy versions of the underlying feature vectors are observed in both the enrollment and query phases. The noisy observations are compressed before being stored in the database, and the user wishes to both identify the correct entry corresponding to the noisy query vector and reconstruct the original feature vector within a desired distortion level. A fundamental capacity-storage-distortion tradeoff is identified for this system in the form of single-letter information theoretic expressions. The relation of this problem to the classical Wyner-Ziv rate-distortion problem is shown, where the noisy query vector acts as the correlated side information available only in the lossy reconstruction of the feature vector. \ua9 1963-2012 IEEE
A general framework for the optimization of energy harvesting communication systems with battery imperfections
Energy harvesting has emerged as a powerful technology for complementing current battery-powered communication systems in order to extend their lifetime. In this paper a general framework is introduced for the optimization of communication systems in which the transmitter is able to harvest energy from its environment. Assuming that the energy arrival process is known non-causally at the transmitter, the structure of the optimal transmission scheme, which maximizes the amount of transmitted data by a given deadline, is identified. Our framework includes models with continuous energy arrival as well as battery constraints. A battery that suffers from energy leakage is studied further, and the optimal transmission scheme is characterized for a constant leakage rate
P3488Exogenous Nitric Oxide promotes maturation of endothelial adherens junctions and protects against thrombin-induced endothelial barrier failure
P192Opposing effects of cAMP/PKA and cAMP/Epac signalling on in vitro angiogenesis: role of Rho gtpases
P181cAMP-mediated Rac1 activation regulates the re-establishment of endothelial adherens junctions and barrier restoration during inflammation
P552Role of PI3K/Akt and MEK/ERK signalling in Epac-mediated endothelial barrier stabilisation and survival
P346Effect of pharmacological activation of adenosine monophosphate activated kinase (AMPK) on endothelial barrier function, proliferation, and angiogenesis
P551Effects of interferon gamma on endothelial barrier function: differential role of classical and non-classical pathways
Crescentic glomerulonephritis associated with nail-patella syndrome in a 13-year-old girl
Nail-patella syndrome (NPS), also known as hereditary osteoonychodysplasia, is a rare autosomal dominant pleiotropic disorder, defined by the association of nail dysplasia, skeletal abnormalities and renal lesions.(1) Skeletal features include absent or hypoplastic patella, patella dislocation, elbow abnormalities (like knee abnormalities), and iliac horns on X-ray. Renal involvement is considered the most serious component and the cause of lethality of this syndrome. Renal involvement may lead to renal failure.(2) The main pathology involves a defect in the glomerular basement membrane (GBM), which has irregular thickening containing electron-lucent areas. The first sign of renal involvement is usually proteinuria, with or without haematuria.(3) Previous studies have estimated that renal involvement occurred in 12-55% of patients with NPS,(4) and renal failure in 5-14% of NPS patients.(5
Crescentic glomerulonephritis associated with nail-patella syndrome in a 13-year-old girl
Nail-patella syndrome (NPS), also known as hereditary osteoonychodysplasia, is a rare autosomal dominant pleiotropic disorder, defined by the association of nail dysplasia, skeletal abnormalities and renal lesions.(1) Skeletal features include absent or hypoplastic patella, patella dislocation, elbow abnormalities (like knee abnormalities), and iliac horns on X-ray. Renal involvement is considered the most serious component and the cause of lethality of this syndrome. Renal involvement may lead to renal failure.(2) The main pathology involves a defect in the glomerular basement membrane (GBM), which has irregular thickening containing electron-lucent areas. The first sign of renal involvement is usually proteinuria, with or without haematuria.(3) Previous studies have estimated that renal involvement occurred in 12-55% of patients with NPS,(4) and renal failure in 5-14% of NPS patients.(5
