37 research outputs found
L-Selectin (CD62L) drives development and progression of non-alcoholic steatohepatitis (NASH) in two dietary mouse models
Full text linkHepatic Nrf2 overexpression inhibits the deleterious effects induced by c-met deficiency in the progression of NASH
Full text linkHepatocyte specific deletion of Mesenchymal-epithelial transition factor (c-Met) leads to the development of severe non-alcoholic-steatohepatitis (NASH) in a murine NASH model
Full text linkβ7-Integrins and MAdCAM-1 play an opposing role during the development of Non-alcoholic steatohepatitis (NASH)
Full text linkThe platelet-derived chemokine CXCL4 exerts protective role in non-alcoholic steatohepatitis (NASH) in vivo
Full text linkc-met vermeidet die Entwicklung Nicht- Alkoholischer- Fettleberhepatitis (NASH) im Mausmodell
Full text linkThe IL-6/GP130 signaling pathway in myeloid cells modulates the inflammatory response during sepsis
Full text linkGp130 Signaling Promotes Development of Acute Experimental Colitis by Facilitating Early Neutrophil/Macrophage Recruitment and Activation
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