Immunoglobulin allotypes Gm and Km in chronic schizophrenia: no apparent association

SynopsisThe distribution of the immunoglobulin allotypic markers G1m(1), G1m(2), G1m(3), G3m(5) and Km(1) was determined in 168 hospitalized Caucasian chronic schizophrenic patients and compared with that in healthy controls. No association between Gm or Km phenotypes and chronic schizophrenia is apparent.</jats:p

Some aspects of human variation in growth and dermatoglyphics

Typescript (roneo copy)Thesis (MSc) -- University of Melbourne, Department of Science, 1969Includes bibliographical referencesUsers are advised that this Aboriginal and/or Torres Strait Islander material may contain culturally sensitive imagery and descriptions which may not normally be used in certain public or community contexts. Annotation and terminology which reflects the creator's attitude or that of the era in which the item was created may be considered inappropriate today. This material may also contain images, voices or names of deceased personsMeans and standard deviations of pattern intensity index and total ridge-count are presented for a limited sample of full-blood Western Australian Aborigines from a mission station near Wiluna. The results are compared with those found by previous workers

Selective Forces and the Maintenance of Immunoglobulin Polymorphisms

In an attempt to identify the role of selective pressures in the maintenance of immunoglobulin polymorphisms, the phenotypes identified by the immunoglobulin allotypes Glm(l), Glm(2), Glm(3), G3m(5) and Km(l) have been determined in a large sample of adult Caucasians resident in Melbourne, Australia. While no evidence for age or sex-related differences was found, non-random distribution of Gm and Km phenotypes suggests the existence of selective forces favoring Gm heterozygotes who are Km (1 + ) and Gm homozygotes who are Km(l —)