7,842 research outputs found

    Sensitive Chemical Compass Assisted by Quantum Criticality

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    The radical-pair-based chemical reaction could be used by birds for the navigation via the geomagnetic direction. An inherent physical mechanism is that the quantum coherent transition from a singlet state to triplet states of the radical pair could response to the weak magnetic field and be sensitive to the direction of such a field and then results in different photopigments in the avian eyes to be sensed. Here, we propose a quantum bionic setup for the ultra-sensitive probe of a weak magnetic field based on the quantum phase transition of the environments of the two electrons in the radical pair. We prove that the yield of the chemical products via the recombination from the singlet state is determined by the Loschmidt echo of the environments with interacting nuclear spins. Thus quantum criticality of environments could enhance the sensitivity of the detection of the weak magnetic field.Comment: 4 pages, 3 figure

    Trapped interacting two-component bosons

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    In this paper we solve one dimensional trapped SU(2) bosons with repulsive δ\delta-function interaction by means of Bethe-ansatz method. The features of ground state and low-lying excited states are studied by numerical and analytic methods. We show that the ground state is an isospin "ferromagnetic" state which differs from spin-1/2 fermions system. There exist three quasi-particles in the excitation spectra, and both holon-antiholon and holon-isospinon excitations are gapless for large systems. The thermodynamics equilibrium of the system at finite temperature is studied by thermodynamic Bethe ansatz. The thermodynamic quantities, such as specific heat etc. are obtained for the case of strong coupling limit.Comment: 15 pages, 9 figure

    Finite-Temperature Scaling of Magnetic Susceptibility and Geometric Phase in the XY Spin Chain

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    We study the magnetic susceptibility of 1D quantum XY model, and show that when the temperature approaches zero, the magnetic susceptibility exhibits the finite-temperature scaling behavior. This scaling behavior of the magnetic susceptibility in 1D quantum XY model, due to the quantum-classical mapping, can be easily experimentally tested. Furthermore, the universality in the critical properties of the magnetic susceptibility in quantum XY model is verified. Our study also reveals the close relation between the magnetic susceptibility and the geometric phase in some spin systems, where the quantum phase transitions are driven by an external magnetic field.Comment: 6 pages, 4 figures, get accepted for publication by J. Phys. A: Math. Theo

    Multi-Player and Multi-Choice Quantum Game

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    We investigate a multi-player and multi-choice quantum game. We start from two-player and two-choice game and the result is better than its classical version. Then we extend it to N-player and N-choice cases. In the quantum domain, we provide a strategy with which players can always avoid the worst outcome. Also, by changing the value of the parameter of the initial state, the probabilities for players to obtain the best payoff will be much higher that in its classical version.Comment: 4 pages, 1 figur

    Dynamics of lattice spins as a model of arrhythmia

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    We consider evolution of initial disturbances in spatially extended systems with autonomous rhythmic activity, such as the heart. We consider the case when the activity is stable with respect to very smooth (changing little across the medium) disturbances and construct lattice models for description of not-so-smooth disturbances, in particular, topological defects; these models are modifications of the diffusive XY model. We find that when the activity on each lattice site is very rigid in maintaining its form, the topological defects - vortices or spirals - nucleate a transition to a disordered, turbulent state.Comment: 17 pages, revtex, 3 figure

    Crystalline Assemblies and Densest Packings of a Family of Truncated Tetrahedra and the Role of Directional Entropic Forces

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    Polyhedra and their arrangements have intrigued humankind since the ancient Greeks and are today important motifs in condensed matter, with application to many classes of liquids and solids. Yet, little is known about the thermodynamically stable phases of polyhedrally-shaped building blocks, such as faceted nanoparticles and colloids. Although hard particles are known to organize due to entropy alone, and some unusual phases are reported in the literature, the role of entropic forces in connection with polyhedral shape is not well understood. Here, we study thermodynamic self-assembly of a family of truncated tetrahedra and report several atomic crystal isostructures, including diamond, {\beta}-tin, and high- pressure lithium, as the polyhedron shape varies from tetrahedral to octahedral. We compare our findings with the densest packings of the truncated tetrahedron family obtained by numerical compression and report a new space filling polyhedron, which has been overlooked in previous searches. Interestingly, the self-assembled structures differ from the densest packings. We show that the self-assembled crystal structures can be understood as a tendency for polyhedra to maximize face-to-face alignment, which can be generalized as directional entropic forces.Comment: Article + supplementary information. 23 pages, 10 figures, 2 table

    Decay of Loschmidt Echo Enhanced by Quantum Criticality

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    We study the transition of a quantum system SS from a pure state to a mixed one, which is induced by the quantum criticality of the surrounding system EE coupled to it. To characterize this transition quantitatively, we carefully examine the behavior of the Loschmidt echo (LE) of EE modelled as an Ising model in a transverse field, which behaves as a measuring apparatus in quantum measurement. It is found that the quantum critical behavior of EE strongly affects its capability of enhancing the decay of LE: near the critical value of the transverse field entailing the happening of quantum phase transition, the off-diagonal elements of the reduced density matrix describing SS vanish sharply.Comment: 4 pages, 3 figure

    Loss of PLA2G6 leads to elevated mitochondrial lipid peroxidation and mitochondrial dysfunction

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    © 2015 The Author.The PLA2G6 gene encodes a group VIA calcium-independent phospholipase A2 beta enzyme that selectively hydrolyses glycerophospholipids to release free fatty acids. Mutations in PLA2G6 have been associated with disorders such as infantile neuroaxonal dystrophy, neurodegeneration with brain iron accumulation type II and Karak syndrome. More recently, PLA2G6 was identified as the causative gene in a subgroup of patients with autosomal recessive early-onset dystonia-Parkinsonism. Neuropathological examination revealed widespread Lewy body pathology and the accumulation of hyperphosphorylated tau, supporting a link between PLA2G6 mutations and Parkinsonian disorders. Here we show that knockout of the Drosophila homologue of the PLA2G6 gene, iPLA2-VIA, results in reduced survival, locomotor deficits and organismal hypersensitivity to oxidative stress. Furthermore, we demonstrate that loss of iPLA2-VIA function leads to a number of mitochondrial abnormalities, including mitochondrial respiratory chain dysfunction, reduced ATP synthesis and abnormal mitochondrial morphology. Moreover, we show that loss of iPLA2-VIA is strongly associated with increased lipid peroxidation levels. We confirmed our findings using cultured fibroblasts taken from two patients with mutations in the PLA2G6 gene. Similar abnormalities were seen including elevated mitochondrial lipid peroxidation and mitochondrial membrane defects, as well as raised levels of cytoplasmic and mitochondrial reactive oxygen species. Finally, we demonstrated that deuterated polyunsaturated fatty acids, which inhibit lipid peroxidation, were able to partially rescue the locomotor abnormalities seen in aged flies lacking iPLA2-VIA gene function, and restore mitochondrial membrane potential in fibroblasts from patients with PLA2G6 mutations. Taken together, our findings demonstrate that loss of normal PLA2G6 gene activity leads to lipid peroxidation, mitochondrial dysfunction and subsequent mitochondrial membrane abnormalities. Furthermore we show that the iPLA2-VIA knockout fly model provides a useful platform for the further study of PLA2G6-associated neurodegeneration
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