Authorship Attribution Using a Neural Network Language Model

In practice, training language models for individual authors is often expensive because of limited data resources. In such cases, Neural Network Language Models (NNLMs), generally outperform the traditional non-parametric N-gram models. Here we investigate the performance of a feed-forward NNLM on an authorship attribution problem, with moderate author set size and relatively limited data. We also consider how the text topics impact performance. Compared with a well-constructed N-gram baseline method with Kneser-Ney smoothing, the proposed method achieves nearly 2:5% reduction in perplexity and increases author classification accuracy by 3:43% on average, given as few as 5 test sentences. The performance is very competitive with the state of the art in terms of accuracy and demand on test data. The source code, preprocessed datasets, a detailed description of the methodology and results are available at https://github.com/zge/authorship-attribution.Comment: Proceedings of the 30th AAAI Conference on Artificial Intelligence (AAAI'16

Stress and productivity patterns of interrupted, synergistic, and antagonistic office activities.

We describe a controlled experiment, aiming to study productivity and stress effects of email interruptions and activity interactions in the modern office. The measurement set includes multimodal data for n = 63 knowledge workers who volunteered for this experiment and were randomly assigned into four groups: (G1/G2) Batch email interruptions with/without exogenous stress. (G3/G4) Continual email interruptions with/without exogenous stress. To provide context, the experiment's email treatments were surrounded by typical office tasks. The captured variables include physiological indicators of stress, measures of report writing quality and keystroke dynamics, as well as psychometric scores and biographic information detailing participants' profiles. Investigations powered by this dataset are expected to lead to personalized recommendations for handling email interruptions and a deeper understanding of synergistic and antagonistic office activities. Given the centrality of email in the modern office, and the importance of office work to people's lives and the economy, the present data have a valuable role to play

Vanadium(v) phenolate complexes for ring opening homo- and co-polymerisation of ε-caprolactone, L-lactide and rac-lactide

The vanadyl complexes [VO(OtBu)L¹ ] (1) and {[VO(OiPr)]₂ (μ-p-L²ᵖ)} (2) {[VO(OR)]₂ (μ-p-L²ᵐ )} (R = iPr 3, tBu 4) have been prepared from [VO(OR)₃ ] (R = nPr, iPr or tBu) and the respective phenol, namely 2,2′-ethylidenebis(4,6-di-tert-butylphenol) (L¹ H₂ ) or α,α,α′,α′-tetra(3,5-di-tert-butyl-2-hydroxyphenyl–p/m-)xylene-para-tetraphenol (L2p/mH₄). For comparative studies, the known complexes [VO(μ-OnPr)L¹]₂ (I), [VOL³ ]₂ (II) (L³H₃ = 2,6-bis(3,5-di-tert-butyl-2-hydroxybenzyl)-4-tert-butylphenol) were prepared. An imido complex {[VCl(Np-tolyl)(NCMe)]₂(μ-p-L²ᵖ)} (5) has been prepared following work-up from [V(Np-tolyl)Cl₃ ], L²ᵖH₄ and Et₃ N. The molecular structures of complexes 1–5 are reported. Complexes 1–5 and I and II have been screened for their ability to ring open polymerise ε-caprolactone, L-lactide or rac-lactide with and without solvent present. The co-polymerization of ε-caprolactone with L-lactide or rac-lactide afforded co-polymers with low lactide content; the reverse addition was ineffective

Short term doxycycline treatment induces sustained improvement in myocardial infarction border zone contractility.

Decreased contractility in the non-ischemic border zone surrounding a MI is in part due to degradation of cardiomyocyte sarcomeric components by intracellular matrix metalloproteinase-2 (MMP-2). We recently reported that MMP-2 levels were increased in the border zone after a MI and that treatment with doxycycline for two weeks after MI was associated with normalization of MMP-2 levels and improvement in ex-vivo contractile protein developed force in the myocardial border zone. The purpose of the current study was to determine if there is a sustained effect of short term treatment with doxycycline (Dox) on border zone function in a large animal model of antero-apical myocardial infarction (MI). Antero-apical MI was created in 14 sheep. Seven sheep received doxycycline 0.8 mg/kg/hr IV for two weeks. Cardiac MRI was performed two weeks before, and then two and six weeks after MI. Two sheep died prior to MRI at six weeks from surgical/anesthesia-related causes. The remaining 12 sheep completed the protocol. Doxycycline induced a sustained reduction in intracellular MMP-2 by Western blot (3649±643 MI+Dox vs 9236±114 MI relative intensity; p = 0.0009), an improvement in ex-vivo contractility (65.3±2.0 MI+Dox vs 39.7±0.8 MI mN/mm2; p<0.0001) and an increase in ventricular wall thickness at end-systole 1.0 cm from the infarct edge (12.4±0.6 MI+Dox vs 10.0±0.5 MI mm; p = 0.0095). Administration of doxycycline for a limited two week period is associated with a sustained improvement in ex-vivo contractility and an increase in wall thickness at end-systole in the border zone six weeks after MI. These findings were associated with a reduction in intracellular MMP-2 activity