147 research outputs found

    Familial "Giant-Cell Hepatitis" in Infancy

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    Anencephaly

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    Neonatal Intensive Care

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    Familial Juvenile Nephronophthisis

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    The recent article entitled "Familial Juvenile Nephronophthisis" by Mangos, et al. (Pediatrics, 34:337, 1964) is marred by a single unsubstantiated statement in the opening paragraph: "Recognition and study of this disease (hereditary nephritis and deafness) had led to the demonstration of a biochemical defect in affected individuals." The carefully worded and informative work of Schafer, et al. (New Engl. J. Med., 267:51, 1962) neither supports nor openly suggests a cause and effect relationship between defective amino acid metabolism and the syndrome of hereditary renal dysfunction and deafness.</jats:p

    Introduction

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    If Too Much of a Good Thing Is BAD, Is Too Much of a Bad Thing BPD?

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    The report by Poets and Sens1 in this issue clearly shows that raising the criteria required for endotracheal intubation and intermittent mandatory ventilation (ET/IMV) in an entire community population of vulnerable, very low birth weight (VLBW) infants reduces bronchopulmonary dysplasia (BPD). Outcomes in all live-born infants with birth weights of between 500 and 1499 g from the German state of Lower Saxony, cared for in tertiary centers (comparable to level III neonatal intensive care units [NICUs] in this country) as well as in community centers (level II NICUs), from 1992 through 1994 were examined. No apparent price was paid for this improvement in pulmonary outcome.</jats:p

    Amniocentesis

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    Monitoring, Birth Defects and Environment: The Problem of Surveillance.

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