Social Norm, Costly Punishment and the Evolution to Cooperation

Both laboratory and field evidence suggest that people tend to voluntarily incur costs to punish non-cooperators. While costly punishment typically reduces the average payoff as well as promotes cooperation. Why does the costly punishment evolve? We study the role of punishment in cooperation promotion within a two-level evolution framework of individual strategies and social norms. In a population with certain social norm, players update their strategies according to the payoff differences among different strategies. In a longer horizon, the evolution of social norm may be driven by the average payoffs of all members of the society. Norms differ in whether they allow or do not allow for the punishment action as part of strategies, and, for the former, they further differ in whether they encourage or do not encourage the punishment action. The strategy dynamics are articulated under different social norms. It is found that costly punishment does contribute to the evolution toward cooperation. Not only does the attraction basin of cooperative evolutionary stable state (CESS) become larger, but also the convergence speed to CESS is faster. These two properties are further enhanced if the punishment action is encouraged by the social norm. This model can be used to explain the widespread existence of costly punishment in human society.social norm; costly punishment; cooperative evolutionary stable state; attraction basin; convergence speed

Worldwide genetic variation of the IGHV and TRBV immune receptor gene families in humans.

The immunoglobulin heavy variable (IGHV) and T cell beta variable (TRBV) loci are among the most complex and variable regions in the human genome. Generated through a process of gene duplication/deletion and diversification, these loci can vary extensively between individuals in copy number and contain genes that are highly similar, making their analysis technically challenging. Here, we present a comprehensive study of the functional gene segments in the IGHV and TRBV loci, quantifying their copy number and single-nucleotide variation in a globally diverse sample of 109 (IGHV) and 286 (TRBV) humans from over a 100 populations. We find that the IGHV and TRBV gene families exhibit starkly different patterns of variation. In addition to providing insight into the different evolutionary paths of the IGHV and TRBV loci, our results are also important to the adaptive immune repertoire sequencing community, where the lack of frequencies of common alleles and copy number variants is hampering existing analytical pipelines

Deformed brane with finite extra dimension

We construct a deformed brane solution generated by a double-kink scalar field and a dilaton scalar field. In this brane scenario the extra dimension is finite, which is due to the introduction of the dilaton field with special form. The finity of the extra dimension will result in the localization of the zero mode for the vector fields. While the localization of the Kalb-Ramond fields {depends} on the coupling to the dilaton. For the fermion fields, {with different values of the dilaton-fermion coupling constant}, there are three types of the effective potential for the fermion KK modes. Moreover, we investigate the effect of the deformation of the brane on the localization, and find that the number of the resonances will increase with the distances of the two sub-branes.Comment: 9 pages, 3 figures, to be published in PR