Servidor de datos y página web para el aprendizaje de SIG en la ingeniería forestal

Los Sistemas de Información geográfica (SIG) son una herramienta de trabajo habitual en el ámbito de la ingeniería forestal, tanto en la faceta de redacción de proyectos, como en la investigación sobre el medio ambiente y el territorio. Cada vez hay más información cartográfica disponible desde servidores de diferentes instituciones, por lo que consideramos que es muy útil contar con una herramienta de organización de la información. En el trabajo que se presenta, se pretende proporcionar a los estudiantes e investigadores en materia forestal un portal que contenga información actualizada y ordenada sobre los recursos existentes compatibles con los SIG. Por tanto constituiráuna herramienta de apoyo que facilitarála fase de documentación, búsqueda de datos compatibles y aprendizaje de las herramientas que sirven de base para el desarrollo de cualquier trabajo técnico o de investigación relacionado con el medio ambiente y el territorio que se apoye en los SIG

Using neuroscience evidence to train pre-service physics teachers on the concepts of heat and cold

Journal of Physics: Conference Series PAPER • THE FOLLOWING ARTICLE ISOPEN ACCESS Using neuroscience evidence to train pre-service physics teachers on the concepts of heat and cold A Ezquerra1 and I Ezquerra-Romano2 Published under licence by IOP Publishing Ltd Journal of Physics: Conference Series, Volume 1287, GIREP-MPTL 2018 9–13 July 2018, San Sebastian, Spain Citation A Ezquerra and I Ezquerra-Romano 2019 J. Phys.: Conf. Ser. 1287 012038 DownloadArticle PDF References Download PDF 150 Total downloads 11 citation on Dimensions. Turn on MathJax Share this article Share this content via email Share on Facebook (opens new window) Share on Twitter (opens new window) Share on Mendeley (opens new window) Hide article information Author affiliations 1 Department of Didactics of Experimental Sciences, Social Sciences and Mathematics. Faculty of Education. Universidad Complutense Madrid, Spain 2 Institute of Cognitive Neuroscience, University College London, UK DOI https://doi.org/10.1088/1742-6596/1287/1/012038 Buy this article in print Sign up for new issue notifications Create citation alert Abstract Teachers must identify, manage and modify alternative conceptions in their students. However, research consistently highlights that future teachers prioritise disciplines' requirements rather than the educational needs of children. Moreover, some researchers have proposed that the way our senses work shapes the development of children's ideas. Neuroscientists have only recently started to understand how the senses transduce and transmit stimuli. In this study, we show an innovative initial training course of Didactics of Physics, which includes the latest understanding of the thermosensory system. We analysed the progression of the educational proposals written by future teachers. This analysis revealed the effect of integrating neuroscience contents in the initial training. We found that the neuroscience insights helped future teachers incorporating children's misconceptions in their proposals. Additionally, they lowered their expectations and became less frustrated. They understood that it is not expected to quickly modify pupils' alternative conceptions because they have a physiological component

Highway to thermosensation: a traced review, from the proteins to the brain

Temperature maintenance and detection are essential for the survival and perpetuation of any species. This review is focused on thermosensation; thus a detailed and traced explanation of the anatomical and physiological characteristics of each component of this sensation is given. First, the proteins that react to temperature changes are identified; next, the nature of the neurons involved in thermosensation is described; and then, the pathways from the skin through the spinal cord to the brain are outlined. Finally, the areas of the brain and their interconnections where thermoperception arises are explained. Transduction of the external and internal temperature information is essentially mediated by the transient receptor potential ion channels (TRPs). These proteins are embedded in the neurons’ membrane and they hyper- or de-polarize neurons in function of the intrinsic voltage and the temperature changes. There are distinct TRP sensors for different temperature ranges. Interestingly, the primary afferent neurons have either cold or hot receptors, so they are dedicated separately to cold or hot sensation. The information is transmitted by different pathways from the skin to the brain, where it either remains separated or is integrated to generate a response. It seems that both the determination of how thermoperception is produced and how we interact with the world are dependent on the particular arrangement and nature of the components, the way of transduction of information and the communication between these elements

From Thermosensation to the Concepts of Heat and Temperature: A Possible Neuroscientific Component

Alternative conceptions in physics are ideas held by people regardless of their age, ability, sex, race and religion. The persistence and universality of these misconceptions suggest that there must be a common underlying factor found in all human beings. In this work, we suggest how the structure and arrangement of our thermosensory system shapes and constrains the creation of the concepts of heat and temperature. Firstly, we outline the main characteristics of alternative conceptions in physics. Then, we describe the neurobiology of thermosensation. The proteins sensitive to temperature changes can be classified as hot- and cold-sensitive. The nervous system maintains mostly this separation in hot- and cold-fibres and thermal information is integrated in specific areas of the central nervous system. Therefore, it seems that the neurobiological structure predisposes us to categorise stimuli into hot and cold. Understanding the relationship between alternative conceptions and the structure of the nervous system can improve the abilities of teachers to deal with students’ ideas. In particular, this knowledge could decrease the frustration of teachers, since they would understand that human physiology is a determinant factor. Therefore, they should not expect to easily modify their students’ alternative conceptions

Gender differences in cheating: loss vs gain framing

We use the die-paradigm to study gender differences in cheating behavior. We find that i) both males and females do not cheat in the absence of financial incentives, ii) both males and females cheat (but not maximally) if reports are associated with financial gains or losses, and iii) males and females do not cheat differentially

Spinodal-assisted crystallization in polymer melts

Recent experiments in some polymer melts quenched below the melting temperature have reported spinodal kinetics in small-angle x-ray scattering before the emergence of a crystalline structure. To explain these observations we propose that the coupling between density and chain conformation induces a liquid-liquid binodal within the equilibrium liquid-crystalline solid coexistence region. A simple phenomenological theory is developed to illustrate this idea, and several experimentally testable consequences are discussed. Shear is shown to enhance the kinetic role of the hidden binodal

Cheating, incentives, and money manipulation

We use different incentive schemes to study truth-telling in a die-roll task when people are asked to reveal the number rolled privately. We find no significant evidence of cheating when there are no financial incentives associated with the reports, but do find evidence of such when the reports determine financial gains or losses (in different treatments). We find no evidence of loss aversion in the standard case in which subjects receive their earnings in a sealed envelope at the end of the session. When subjects manipulate the possible earnings, we find evidence of less cheating, particularly in the loss setting; in fact, there is no significant difference in behavior between the non-incentivized case and the loss setting with money manipulation. We interpret our findings in terms of the moral cost of cheating and differences in the perceived trust and beliefs in the gain and the loss frames

Parkin loss of function contributes to RTP801 elevation and neurodegeneration in Parkinson"s disease

Mutations in the PARK2 gene are associated with an autosomal recessive form of juvenile parkinsonism (AR-JP). These mutations affect parkin solubility and impair its E3 ligase activity, leading to a toxic accumulation of proteins within susceptible neurons that results in a slow but progressive neuronal degeneration and cell death. Here, we report that RTP801/REDD1, a pro-apoptotic negative regulator of survival kinases mTOR and Akt, is one of such parkin substrates. We observed that parkin knockdown elevated RTP801 in sympathetic neurons and neuronal PC12 cells, whereas ectopic parkin enhanced RTP801 poly-ubiquitination and proteasomal degradation. In parkin knockout mouse brains and in human fibroblasts from AR-JP patients with parkin mutations, RTP801 levels were elevated. Moreover, in human postmortem PD brains with mutated parkin, nigral neurons were highly positive for RTP801. Further consistent with the idea that RTP801 is a substrate for parkin, the two endogenous proteins interacted in reciprocal co-immunoprecipitates of cell lysates. A potential physiological role for parkin-mediated RTP801 degradation is indicated by observations that parkin protects neuronal cells from death caused by RTP801 overexpression by mediating its degradation, whereas parkin knockdown exacerbates such death. Similarly, parkin knockdown enhanced RTP801 induction in neuronal cells exposed to the Parkinson's disease mimetic 6-hydroxydopamine and increased sensitivity to this toxin. This response to parkin loss of function appeared to be mediated by RTP801 as it was abolished by RTP801 knockdown. Taken together these results indicate that RTP801 is a novel parkin substrate that may contribute to neurodegeneration caused by loss of parkin expression or activity