Growth and electronic and magnetic structure of iron oxide films on Pt(111)

Ultrathin (111)-oriented polar iron oxide films were grown on a Pt(111) single crystal either by the reactive deposition of iron or oxidation of metallic iron monolayers. These films were characterized using low energy electron diffraction, scanning tunneling microscopy and conversion electron Mossbauer spectroscopy. The reactive deposition of Fe led to the island growth of Fe3O4, in which the electronic and magnetic properties of the bulk material were modulated by superparamagnetic size effects for thicknesses below 2 nm, revealing specific surface and interface features. In contrast, the oxide films with FeO stoichiometry, which could be stabilized as thick as 4 nm under special preparation conditions, had electronic and magnetic properties that were very different from their bulk counterpart, w\"ustite. Unusual long range magnetic order appeared at room temperature for thicknesses between three and ten monolayers, the appearance of which requires severe structural modification from the rock-salt structure.Comment: 17 pages, 6 figures, 50 reference

A Demonstration Experiment for the Forecast of Magnetic Field and Field Errors in the Large Hadron Collider

In order to reduce the burden on the beam-based feedback, the Large Hadron Collider control system is equipped with the Field Description for the LHC (FiDeL) which provides a forecast of the magnetic field and the multipole field errors. FiDeL has recently been extensively tested at CERN to determine main field tracking, multipole forecasting and compensation accuracy. This paper describes the rationale behind the tests, the procedures employed to power the main magnets and their correctors, and finally, we present the results obtained. We also give an indication of the prediction accuracy that the system can deliver during the operation of the LHC and we discuss the implications that these will have on the machine performance

Main Field Tracking Measurement in the LHC Superconducting Dipole and Quadrupole Magnets

One of the most stringent requirements during the energy ramp of the Large Hadron Collider (LHC) is to have a constant ratio between dipole-quadrupole and dipole-dipole field so as to control the variation of the betatron tune and of the beam orbit throughout the acceleration phase, hence avoiding particle loss. To achieve the nominal performance of the LHC, a maximum variation of ±0.003 tune units can be tolerated. For the commissioning with low intensity beams, acceptable bounds are up to 30 times higher. For the quadrupole-dipole integrated field ratio, the above requirements translate in the tight windows of 6 ppm and 180 ppm, while for dipole differences between sectors the acceptable error is of the order of 10^-4. Measurement and control at this level are challenging. For this reason we have launched a dedicated measurement R&D to demonstrate that these ratios can be measured and controlled within the limits for machine operation. In this paper we present the techniques developed to power the magnets during the current ramps, the instrumentation and data acquisition setup used to perform the tracking experiments, the calibration procedure and the data reduction employed

A Demonstration Experiment for the Main Field Tracking and the Sextupole and Decapole Compensation in the LHC Main Magnets

A dedicated measurement campaign was set up to test the FiDeL concept and its LSA implementation. The test was performed by demonstrating the tracking of B1 and B2 for two LHC main dipoles and one LHC main quadrupole. It also included the compensation of the b3 and b5 harmonics in the dipole magnets using the sextupole and decapole corrector magnets. In this report we present the techniques developed to power the magnets for these tests during the current ramps; the instrumentation and data acquisition setup used to perform the tracking experiments; the calibration procedure and data corrections employed; and finally the main results obtained

The LHC Injection Tests

A series of LHC injection tests was performed in August and September 2008. The first saw beam injected into sector 23; the second into sectors 78 and 23; the third into sectors 78-67 and sectors 23-34-45. The fourth, into sectors 23-34-45, was performed the evening before the extended injection test on the 10th September which saw both beams brought around the full circumference of the LHC. The tests enabled the testing and debugging of a number of critical control and hardware systems; testing and validation of instrumentation with beam for the first time; deployment, and validation of a number of measurement procedures. Beam based measurements revealed a number of machine configuration issues that were rapidly resolved. The tests were undoubtedly an essential precursor to the successful start of LHC beam commissioning. This paper provides an outline of preparation for the tests, the machine configuration and summarizes the measurements made and individual system performance

Cellular Radiosensitivity: How much better do we understand it?

Purpose: Ionizing radiation exposure gives rise to a variety of lesions in DNA that result in genetic instability and potentially tumorigenesis or cell death. Radiation extends its effects on DNA by direct interaction or by radiolysis of H2O that generates free radicals or aqueous electrons capable of interacting with and causing indirect damage to DNA. While the various lesions arising in DNA after radiation exposure can contribute to the mutagenising effects of this agent, the potentially most damaging lesion is the DNA double strand break (DSB) that contributes to genome instability and/or cell death. Thus in many cases failure to recognise and/or repair this lesion determines the radiosensitivity status of the cell. DNA repair mechanisms including homologous recombination (HR) and non-homologous end-joining (NHEJ) have evolved to protect cells against DNA DSB. Mutations in proteins that constitute these repair pathways are characterised by radiosensitivity and genome instability. Defects in a number of these proteins also give rise to genetic disorders that feature not only genetic instability but also immunodeficiency, cancer predisposition, neurodegeneration and other pathologies. Conclusions: In the past fifty years our understanding of the cellular response to radiation damage has advanced enormously with insight being gained from a wide range of approaches extending from more basic early studies to the sophisticated approaches used today. In this review we discuss our current understanding of the impact of radiation on the cell and the organism gained from the array of past and present studies and attempt to provide an explanation for what it is that determines the response to radiation