1,539 research outputs found
Effect of winding harmonics on the asynchronous torque of a single-phase line start permanant-magnet motor
This paper presents an analytical method for calculating the effect of winding harmonics on the asynchronous torque of a single-phase line-start permanent-magnet motor. The method is an extension of earlier work, which combines symmetrical-component analysis with dq-axis theory to model the various components of forward and backward rotating fields. The effect of individual winding harmonics is brought out both theoretically and experimentally, by comparing calculated and measured torque/speed characteristics for a series of six motors with different distributions of turns in both the main and auxiliary windings
Mandate-driven networking eco-system : a paradigm shift in end-to-end communications
The wireless industry is driven by key stakeholders that follow a holistic approach of "one-system-fits-all" that leads to moving network functionality of meeting stringent End-to-End (E2E) communication requirements towards the core and cloud infrastructures. This trend is limiting smaller and new players for bringing in new and novel solutions. For meeting these E2E requirements, tenants and end-users need to be active players for bringing their needs and innovations. Driving E2E communication not only in terms of quality of service (QoS) but also overall carbon footprint and spectrum efficiency from one specific community may lead to undesirable simplifications and a higher level of abstraction of other network segments may lead to sub-optimal operations. Based on this, the paper presents a paradigm shift that will enlarge the role of wireless innovation at academia, Small and Medium-sized Enterprises (SME)'s, industries and start-ups while taking into account decentralized mandate-driven intelligence in E2E communications
Formation energy and interaction of point defects in two-dimensional colloidal crystals
The manipulation of individual colloidal particles using optical tweezers has
allowed vacancies to be created in two-dimensional (2d) colloidal crystals,
with unprecedented possibility of real-time monitoring the dynamics of such
defects (Nature {\bf 413}, 147 (2001)). In this Letter, we employ molecular
dynamics (MD) simulations to calculate the formation energy of single defects
and the binding energy between pairs of defects in a 2d colloidal crystal. In
the light of our results, experimental observations of vacancies could be
explained and then compared to simulation results for the interstitial defects.
We see a remarkable similarity between our results for a 2d colloidal crystal
and the 2d Wigner crystal (Phys. Rev. Lett. {\bf 86}, 492 (2001)). The results
show that the formation energy to create a single interstitial is
lower than that of the vacancy. Because the pair binding energies of the
defects are strongly attractive for short distances, the ground state should
correspond to bound pairs with the interstitial bound pairs being the most
probable.Comment: 5 pages, 2 figure
A unified radio control architecture for prototyping adaptive wireless protocols
Experimental optimization of wireless protocols and validation of novel solutions is often problematic, due to limited configuration space present in commercial wireless interfaces as well as complexity of monolithic driver implementation on SDR-based experimentation platforms. To overcome these limitations a novel software architecture is proposed, called WiSHFUL, devised to allow: i) maximal exploitation of radio functionalities available in current radio chips, and ii) clean separation between the logic for optimizing the radio protocols (i.e. radio control) and the definition of these protocols
Optical constants of magnetron-sputtered magnesium films in the 25-1300 eV energy range
The transmittance of dc magnetron-sputtered Mg thin films was measured in the 25-1300 eV spectral range. Freestanding Mg films protected with Al layers were characterized ex situ. Transmittance measurements were used to obtain the extinction coefficient k of Mg films. The obtained k values along with the data available in the literature, and with interpolations and extrapolations for the rest of the spectrum, were used to obtain the real part of the index of refraction n by the Kramers-Krönig analysis. Sum-rule tests indicated a good consistency of the data. © 2010 American Institute of Physics.Peer Reviewe
Arc controls AMPAR endocytosis through a direct interaction with clathrin-adaptor protein 2
The activity-regulated cytoskeleton-associated (Arc) protein control synaptic strength by facilitating AMPA receptor (AMPAR) endocytosis. Here we demonstrate that Arc targets AMPAR to be internalized through a direct interaction with the clathrin-adaptor protein 2 (AP-2). We show that Arc overexpression overexpression in dissociated hippocampal neurons obtained from C57BL/6 mouse reduces the density of AMPAR GluA1 subunits at the cell surface and reduces the amplitude and rectification of AMPAR-mediated miniature-excitatory postsynaptic currents (mEPSC). Mutations of Arc, that prevent the AP-2 interaction reduce Arc-mediated endocytosis of GluA1 and abolish the reduction in AMPAR-mediated mEPSC amplitude and rectification. Depletion of the AP-2 subunit µ2 blocks the Arc-mediated reduction in mEPSC amplitude, effect that is restored by re-introducing µ2. The Arc/AP-2 interaction plays an important role in homeostatic synaptic scaling as the Arc-dependent decrease in mEPSC amplitude, induced by a chronic increase in neuronal activity, is inhibited by AP-2 depletion. This data provides a mechanism to explain how activity-dependent expression of Arc decisively controls the fate of AMPAR at the cell surface and modulates synaptic strength, via the direct interaction with the endocytic clathrin adaptor AP-2
Directive 77/187: The EEC’s Attempt to Protect Employees Upon the Sale or Transfer of Businesses
A comparative study of high-field diamagnetic fluctuations in deoxygenated YBa2Cu3O(7-x) and polycrystalline (Bi-Pb)2Sr2Ca3O(10)
We studied three single crystals of YBa2Cu3O{7-x} with Tc= 62.5, 52, and 41
K, and a textured specimen of (Bi-Pb)2Sr2Ca2Cu3O10 with Tc=108 K, for H//c
axis. The reversible data were interpreted in terms of 2D lowest-Landau-level
fluctuation theory. The data were fit well by the 2D LLL expression for
magnetization obtained by Tesanovic etal., producing reasonable values of kappa
but larger values of dHc2/dT. Universality was studied by obtaining a
simultaneous scaling of Y123 data and Bi2223. An expression for the 2D x-axis
LLL scaling factor used to obtain the simultaneous scaling was extracted from
theory, and compared with the experimental values. The comparison between the
values of the x-axis produced a deviation of 40% which suggests that the
hypothesis of universality of the 2D-LLL fluctuations is not supported by the
studied samples. We finaly observe that Y123 magnetization data for
temperatures above obbey a universal scaling obtained for the diamagnetic
fluctuation magnetization from a theory considering non-local field effects.
The same scaling was not obbeyed by the corresponding magnetization calculated
from the two-dimensional lowest-Landau-level theory.Comment: 7 pages 5 figures, accept in Journ. Low Temp. Phy
Chronic AMPK activity dysregulation produces myocardial insulin resistance in the human Arg302Gln-PRKAG2 glycogen storage disease mouse model
BACKGROUND: The cardiac PRKAG2 mutation in the γ2-subunit of adenosine monophosphate activated kinase (AMPK) is characterized by excessive glycogen deposition, hypertrophy, frequent arrhythmias, and progressive conduction system disease. We investigated whether myocardial glucose uptake (MGU) was augmented following insulin stimulation in a mouse model of the PRKAG2 cardiac syndrome. METHODS: Myocardial and skeletal muscle glucose uptake was assessed with 2-[(18)F]fluoro-2-deoxyglucose positron emission tomography imaging in n = 3 transgenic wildtype (TGwt) vs n = 7 PRKAG2 mutant (TGmut) mice at baseline and 1 week later, 30 min following acute insulin. Systolic function, cardiac glycogen stores, phospho-AMPK α, and insulin-receptor expression levels were analyzed to corroborate to the in vivo findings. RESULTS: TGmut Patlak Ki was reduced 56% at baseline compared to TGwt (0.3 ± 0.2 vs 0.7 ± 0.1, t test p = 0.01). MGU was augmented 71% in TGwt mice following acute insulin from baseline (0.7 ± 0.1 to 1.2 ± 0.2, t test p < 0.05). No change was observed in TGmut mice. As expected for this cardiac specific transgene, skeletal muscle was unaffected at baseline with a 33% to 38% increase (standard uptake values) for both genotypes following insulin stimulation. TGmut mice had a 47% reduction in systolic function with a fourfold increase in cardiac glycogen stores correlated with a 29% reduction in phospho-AMPK α levels. There was no difference in cardiac insulin receptor expression between mouse genotypes. CONCLUSIONS: These results demonstrate a correlation between insulin resistance and AMPK activity and provide the basis for the use of this animal model for assessing metabolic therapy in the treatment of affected PRKAG2 patients
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