Wartenberg’s migrant sensory neuritis: a prospective follow-up study

Migrant sensory neuropathy (Wartenberg’s migrant sensory neuritis) is characterized by sudden numbness in the distribution of one or multiple cutaneous nerves. To study disease course and outcome, we prospectively followed 12 patients who presented to our tertiary referral neuromuscular outpatient clinic between January 2003 and January 2004. Medical history, neurological, laboratory and electrophysiological examinations were obtained from all patients. All patients were reviewed a second time in 2007, and five had a follow-up electrophysiological examination. At the first visit, 50% described an episode of stretching preceding the sensory complaints. All but three described pain in the affected area before or concomitant with sensory loss. At clinical examination a median of six skin areas were affected, and in 75% this could be confirmed by nerve conduction studies in at least one nerve. Forty-two percent had involvement of the trigeminal nerve. After a mean disease duration of 7.5 years, three patients reported a complete disappearance of sensory complaints and five that the pain had disappeared, but numbness remained. Three patients still had both painful and numb sensory deficits. One patient developed a distal symmetric sensory polyneuropathy. In conclusion, Wartenberg’s sensory neuritis is a distinct, exclusively sensory, neuropathy, marked by pain preceding numbness in affected nerves. An episode of stretching preceding pain is not necessary for the diagnosis. Wartenberg’s sensory neuritis often retains its spotty, exclusively sensory characteristics after long term follow-up

Hospitalization rates among patients with primary Sjögren's syndrome: a population-based study, 1195-2016

International audienc

Hospitalization rates among patients with primary Sjögren's syndrome: a population-based study, 1195-2016

International audienc

Neuropsychiatric Lupus in clinical practice

Systemic lupus erythematosus (SLE) is a chronic autoimmune disease involving multiple organs, characterized by the production of autoantibodies and the development of tissue injury. The etiology of SLE is partially known, involving multiple genetic and environmental factors. As many as 50% of patients with SLE have neurological involvement during the course of their disease. Neurological manifestations are associated with impaired quality of life, and high morbidity and mortality rates. Nineteen neuropsychiatric syndromes have been identified associated with SLE, and can be divided into central and peripheral manifestations. This article reviews major neuropsychiatric manifestations in patients with SLE and discusses their clinical features, radiological findings and treatment options.Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)Univ Fed Sao Paulo, Div Neurol Geral, Dept Neurol & Neurocirurgia, Sao Paulo, SP, BrazilUniv Estadual Ceara, Ctr Ciencias Saude, Fortaleza, CE, BrazilUniv Fed Ceara, Div Neurol, Dept Med Clin, Fac Med, Fortaleza, CE, BrazilUniv Fed Sao Paulo, Divsao Reumatol, Sao Paulo, SP, BrazilUniversidade Federal de São Paulo, Divisão de Neurologia Geral, Departamento de Neurologia e Neurocirurgia, São Paulo SP, BrasilUniversidade Federal de São Paulo, Divisão de Reumatologia, São Paulo SP, BrasilFAPESP: 2015/08833-7Web of Scienc