Hadron and hadron-cluster production in a hydrodynamical model including particle evaporation

We discuss the evolution of the mixed phase at RHIC and SPS within boostinvariant hydrodynamics. In addition to the hydrodynamical expansion, we also consider evaporation of particles off the surface of the fluid. The back-reaction of the evaporation process on the dynamics of the fluid shortens the lifetime of the mixed phase. In our model this lifetime of the mixed phase is <12 fm/c in Au+Au at RHIC and <6.5 fm/c in Pb+Pb at SPS, even in the limit of vanishing transverse expansion velocity. Strangeness separation occurs, especially in events (or at rapidities) with relatively high initial net baryon and strangeness number, enhancing the multiplicity of MEMOs (multiply strange nuclear clusters). If antiquarks and antibaryons reach saturation in the course of the pure QGP or mixed phase, we find that at RHIC the ratio of antideuterons to deuterons may exceed 0.3 and even anti-helium to helium>0.1. Due to fluctuations, at RHIC even negative baryon number at midrapidity is possible in individual events, so that the antibaryon and antibaryon-cluster yields exceed those of the corresponding baryons and clusters.Comment: 17 pages, Latex, epsfig stylefil

Charged particle production in the Pb+Pb system at 158 GeV/c per nucleon

Charged particle multiplicities from high multiplicity central interactions of 158 GeV/nucleon Pb ions with Pb target nuclei have been measured in the central and far forward projectile spectator regions using emulsion chambers. Multiplicities are significantly lower than predicted by Monte Carlo simulations. We examine the shape of the pseudorapidity distribution and its dependence on centrality in detail.Comment: 17 pages text plus 12 figures in postscript 12/23/99 -- Add TeX version of sourc

Ankyrin is the major oxidised protein in erythrocyte membranes from end-stage renal disease patients on chronic haemodialysis and oxidation is decreased by dialysis and vitamin C supplementation

Chronically haemodialysed end-stage renal disease patients are at high risk of morbidity arising from complications of dialysis, the underlying pathology that has led to renal disease and the complex pathology of chronic kidney disease. Anaemia is commonplace and its origins are multifactorial, involving reduced renal erythropoietin production, accumulation of uremic toxins and an increase in erythrocyte fragility. Oxidative damage is a common risk factor in renal disease and its co-morbidities and is known to cause erythrocyte fragility. Therefore, we have investigated the hypothesis that specific erythrocyte membrane proteins are more oxidised in end-stage renal disease patients and that vitamin C supplementation can ameliorate membrane protein oxidation. Eleven patients and 15 control subjects were recruited to the study. Patients were supplemented with 2 × 500 mg vitamin C per day for 4 weeks. Erythrocyte membrane proteins were prepared pre- and post-vitamin C supplementation for determination of protein oxidation. Total protein carbonyls were reduced by vitamin C supplementation but not by dialysis when investigated by enzyme linked immunosorbent assay. Using a western blot to detect oxidised proteins, one protein band, later identified as containing ankyrin, was found to be oxidised in patients but not controls and was reduced significantly by 60% in all patients after dialysis and by 20% after vitamin C treatment pre-dialysis. Ankyrin oxidation analysis may be useful in a stratified medicines approach as a possible marker to identify requirements for intervention in dialysis patients