14,809 research outputs found
Precision teaching and fluency: the effects of charting and goal-setting on skaters’ performance
This research involved three successive studies where precision teaching methods were used to increase roller skaters performance rates of basic skating skills. The first study compared precision teaching methods both with and without the use of charting in a group design, and compared both within-subject and betweensubject differences for 12 skaters. Results showed that charting did not increase performance rates over that seen when charting was not used. The second study was a group design which compared two different types of goals, or performance aims, on skaters‟ performance. No difference was found between the two groups of 5 skaters when one group used a fixed, difficult goal and a second used a flexible, easier goal. In the final study, a single-subject design was used and 8 skaters completed a control condition where no goals were set before a goal was introduced for 4 skaters. It was found that an immediate increase in performance rates occurred following the introduction of the goal. Overall these three studies showed that skaters improved their performance rates over sessions, even in the absence of charting and/or goals, demonstrating that precision teaching can be applied to the sport of roller skating
Personality and religious maturity
A sample of 226 students attending seminaries, theological colleges, and bible schools completed Newton Malony’s Religious Status Inventory (as a measure of religious maturity) alongside the short-form Revised Eysenck Personality Questionnaire. The data demonstrated some ways in which stable toughminded extraverts projected higher levels of religious maturity according to some of Malony’s criteria
Time Zones as Cues for Coordination: Latitude, Longitude, and Letterman
Market productivity is often greater, and leisure and other household activities more enjoyable, when people perform them simultaneously. Beyond pointing out the positive externalities of synchronicity, economists have not attempted to identify exogenous determinants of timing. We develop a theory illustrating conditions under which synchronicity will vary and identify three factors %u2014 the amount of daylight, the timing of television programming, and differences in time zones %u2014 that can alter timing. Using the American Time Use Survey for 2003 and 2004, we first show that an exogenous shock to time in one area due to non-adherence to daylight-saving time leads its residents to alter their work schedules to continue coordinating their activities with those of people elsewhere. With time use data from Australia, we also demonstrate the same response to a similar shock there. We then show that both television timing and the benefits of coordinating across time zones in the U.S. generally affect the timing of market work and sleep, the two most time-consuming activities people undertake. While these impacts do not differ greatly by people's demographic characteristics, workers in industries where we would expect more coordination outside of their local areas are more responsive to the effects of time zones.
Plio-Pleistocene fossil mammalian microfauna of southern Africa - a preliminary report including description of two new fossil Muroid genera (Mammalia: Rodentia)
Analyses are presented of the mammalian component of rich microfaunal fossil breccia collections mainly of owl pellet origin from the Transvaal Plio-Pleistocene australopithecine sites Kromdraai, Sterkfontein and Makapansgat Limeworks, with briefer references to Swartkrans, Langebaanweg and the Makapansgat Cave of Hearths. Identification of rodent incisors has proved useful in showing Cryptomys robertsi to be a distinct extinct species occurring with C. hottentotus, and in indicating, through the common possession of doubly ridged incisors, a relationship between Mystromys , the Cricetomyidae and certain fossil Cricetodontidae. The fossil assemblages are generally similar to modern ones but elephant shrews (several species) and dormice are relatively commoner, and the once dominant cricetid Mystromys has declined in favour of the murid Mastomys. Two genera, Crocidura and Saccostomus are absent from the older fossil sites, appearing only in the more recent Cave of Hearths, while on the other hand there are certain lineages now extinct. Of these Mystromys darti Lavocat has been rediscovered in abundance in in situ Rodent Corner breccia at Makapansgat, yet it is totally absent from other parts of the Limeworks deposit, suggesting a more complex stratigraphy than previously realised. It is referred to a new genus, Stenodontomys, with a second species from Langebaanweg. Another extinct cricetid previously known under a manuscript name as "Mystromys cookei", common to Makapansgat, Taung and the Krugersdorp district sites, is formally described for the first time also under a new generic name, Proodontomys. On microfaunal evidence Makapansgat is definitely older than the Krugersdorp sites, of which Kromdraai is perhaps the oldest and Swartkrans the youngest. Certain extinct fossils link Makapansgat to Langebaanweg (Stenodontomys), Kromdraai (Macroscelides proboscideus vagans) and Taung (Gypsorhychus). Suggestions that Taung is significantly younger than other australopithecine sites are not supported
The effects of retardation on the topological plasmonic chain: plasmonic edge states beyond the quasistatic limit
We study a one-dimensional plasmonic system with non-trivial topology: a
chain of metallic nanoparticles with alternating spacing, which is the
plasmonic analogue to the Su-Schreiffer-Heeger model. We extend previous
efforts by including long range hopping with retardation and radiative damping,
which leads to a non-Hermitian Hamiltonian with frequency dependence. We
calculate band structures numerically and show that topological features such
as quantised Zak phase persist due to chiral symmetry. This predicts parameters
leading to topologically protected edge modes, which allows for positioning of
disorder-robust hotspots at topological interfaces, opening up novel
nanophotonics applications
Stimulation of microglial metabotropic glutamate receptor mGlu2 triggers tumor necrosis factor alpha-induced neurotoxicity in concert with microglial-derived fas ligand
Activated microglia may be detrimental to neuronal survival in a number of neurodegenerative diseases. Thus, strategies that reduce microglial neurotoxicity may have therapeutic benefit. Stimulation of group II metabotropic glutamate (mGlu) receptors on rat primary microglia with the specific group II agonist 2S, 2 ' R, 3 ' R- 2-(2 ', 3 '-dicarboxy-cyclopropyl) glycine for 24 h induced microglial activation and resulted in a neurotoxic microglial phenotype. These effects were attributable to preferential mGlu2 stimulation, because N-acetyl-L-aspartyl-L-glutamate, a specific mGlu3 agonist, did not induce microglial activation or neurotoxicity. Stimulation of microglial mGlu2 but not mGlu3 induced caspase-3 activation in cerebellar granule neurons in culture, using microglial-conditioned media as well as cocultures. Stimulation of microglial mGlu2 induced tumor necrosis factor-alpha(TNF alpha) release, which contributed to microglial neurotoxicity mediated via neuronal TNF receptor 1 and caspase-3 activation. Stimulation of microglial group I or III mGlu receptors did not induce TNF alpha release. TNF alpha was only neurotoxic in the presence of microglia or microglial-conditioned medium. The toxicity of TNF alpha could be prevented by coexposure of neurons to conditioned medium from microglia stimulated by the specific group III agonist L-2-amino-4-phosphono-butyric acid. The neurotoxicity of TNF alpha derived from mGlu2-stimulated microglia was potentiated by microglial-derived Fas ligand (FasL), the death receptor ligand. FasL was constitutively expressed in microglia and shed after mGlu2 stimulation. Our data suggest that selective and inverse modulation of microglial mGlu2 and mGlu3 may prove a therapeutic target in neuroinflammatory diseases such as Alzheimer's disease and multiple sclerosis
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