Crumpling wires in two dimensions

An energy-minimal simulation is proposed to study the patterns and mechanical properties of elastically crumpled wires in two dimensions. We varied the bending rigidity and stretching modulus to measure the energy allocation, size-mass exponent, and the stiffness exponent. The mass exponent is shown to be universal at value $D_{M}=1.33$. We also found that the stiffness exponent $\alpha =-0.25$ is universal, but varies with the plasticity parameters $s$ and $\theta_{p}$. These numerical findings agree excellently with the experimental results

Using LIP to Gloss Over Faces in Single-Stage Face Detection Networks

This work shows that it is possible to fool/attack recent state-of-the-art face detectors which are based on the single-stage networks. Successfully attacking face detectors could be a serious malware vulnerability when deploying a smart surveillance system utilizing face detectors. We show that existing adversarial perturbation methods are not effective to perform such an attack, especially when there are multiple faces in the input image. This is because the adversarial perturbation specifically generated for one face may disrupt the adversarial perturbation for another face. In this paper, we call this problem the Instance Perturbation Interference (IPI) problem. This IPI problem is addressed by studying the relationship between the deep neural network receptive field and the adversarial perturbation. As such, we propose the Localized Instance Perturbation (LIP) that uses adversarial perturbation constrained to the Effective Receptive Field (ERF) of a target to perform the attack. Experiment results show the LIP method massively outperforms existing adversarial perturbation generation methods -- often by a factor of 2 to 10.Comment: to appear ECCV 2018 (accepted version

DLC2 modulates angiogenic responses in vascular endothelial cells by regulating cell attachment and migration.

Deleted in liver cancer 1 (DLC1) is a RhoGTPase activation protein-containing tumor suppressor that associates with various types of cancer. Although DLC2 shares a similar domain structure with that of DLC1, the function of DLC2 is not well characterized. Here, we describe the expression and ablation of DLC2 in mice using a reporter-knockout approach. DLC2 is expressed in several tissues and in endothelial cells (ECs) of blood vessels. Although ECs and blood vessels show no histological abnormalities and mice appear overall healthy, DLC2-mutant mice display enhanced angiogenic responses induced by matrigel and by tumor cells. Silencing of DLC2 in human ECs has reduced cell attachment, increased migration, and tube formation. These changes are rescued by silencing of RhoA, suggesting that the process is RhoA pathway dependent. These results indicate that DLC2 is not required for mouse development and normal vessel formation, but may protect mouse from unwanted angiogenesis induced by, for example, tumor cells